austlii:nsw/NSWPICMP/2025/425
General information, not legal advice.
NSWPICMP
2025-06-18
Original judgment source is linked above.
Youi Pty Limited v Green [2025] NSWPICMP 425
MOTOR ACCIDENTS - Motor Accident Injuries Act 2017; review of Medical Assessment Certificate (MAC); claimant suffered injury in a motor vehicle accident; Medical Assessor (MA) certified injuries to the right knee and head caused by the accident were soft tissue injuries and therefore threshold injuries; he certified injury to the left knee was not a threshold injury; application for review by insurer; MRI following accident showed no acute tear of medial meniscus; acute tear of medial meniscus shown on MRI scan of 14 May 2024; - injury to head was soft tissue injury with no evidence of traumatic brain injury; accident caused soft tissue injury to both knees; acute tear of the medial meniscus of the left knee not caused by accident; soft tissue injury of head and both knees all threshold injuries; MAC revoked.
Certificate issued under s 7.23(1) of the Motor Accident Injuries Act 2017
"[A]n injury to tissue that connects, supports or surrounds other structures or organs of the body (such as muscles, tendons, ligaments, menisci, cartilage, fascia, fibrous tissues, fat, blood vessels and synovial membranes), but not an injury to nerves or a complete or partial rupture of tendons, ligaments, menisci or cartilage."
"5.3 The assessment will determine whether the injury related to the claim is a soft tissue injury or a threshold psychological or psychiatric injury caused by the motor accident.
5.4 Insurers should not require injured persons to undergo diagnostic imaging for the purpose of the insurer determining whether the injury related to the claim is a threshold injury. Diagnostic imaging is not considered necessary to assess threshold injury.
5.5 A diagnosis for the purpose of a threshold injury decision must be based on a clinical assessment by a medical practitioner or other suitably qualified person independent from the insurer.
5.6 The assessment of whether an injury caused by the accident is a threshold injury for the purposes of the Act should be based on the evidence available and include all relevant findings derived from:
(a) a comprehensive accurate history, including pre-accident history and pre-existing conditions
(b) a review of all relevant records available at the assessment
(c) a comprehensive description of the injured person's current symptoms
(d) a careful and thorough physical and/or psychological examination
(e) diagnostic tests available at the assessment. Imaging findings that are used to support the assessment should correspond with symptoms and findings on examination."
"The question of causation of injuries was not dealt with in Part 5 of the Guidelines but causation was addressed in Part 6, which related to assessment of permanent impairment. There is no reason to think that different principles were intended to be applied when a medical assessment was being made in relation to causation of minor injuries. Clauses 6.5 to 6.7 provided:
6.5 An assessment of the degree of permanent impairment is a medical assessment matter under clause 2(a) of Schedule 2 of the Act. The assessment must determine the degree of permanent impairment of the injured person as a result of the injury caused by the motor accident. A determination as to whether the injured person's impairment is related to the accident in question is therefore implied in all such assessments. Medical assessors must be aware of the relevant provisions of the AMA4 Guides, as well as the common law principles that would be applied by a court (or claims assessor) in considering such issues.
6.6 Causation is defined in the Glossary at page 316 of the AMA4 Guides as follows:
'Causation means that a physical, chemical or biologic factor contributed to the occurrence of a medical condition. To decide that a factor alleged to have caused or contributed to the occurrence or worsening of a medical condition has, in fact, done so, it is necessary to verify both of the following:
This, therefore, involves a medical decision and a non-medical informed judgement.
6.7 There is no simple common test of causation that is applicable to all cases, but the accepted approach involves determining whether the injury (and the associated impairment) was caused or materially contributed to by the motor accident. The motor accident does not have to be a sole cause as long as it is a contributing cause, which is more than negligible. Considering the question 'Would this injury (or impairment) have occurred if not for the accident?' may be useful in some cases, although this is not a definitive test and may be inapplicable in circumstances where there are multiple contributing causes."
(a) the imaging report relating to the MRI scan of 4 November 2011;
(b) clinical notes of Burraneer Family Practice including the clinical notes of Dr Emanuel Papapetros and Dr Kylie Partlin relating to treatment of the claimant in the period two years pre accident to date, and
(c) clinical notes of the physiotherapist Mr Travis Adelerhof for the period 1 January 2024 to date.
Achilles tendon repair in 1995, a right medial meniscectomy in 1998 and another Achilles tendon repair on the other side in 2005. 5. Medical Assessor Cameron reported Ms Green said she did not lose consciousness, she had a "hysterical reaction", and she "froze". He reported she was assessed at Sutherland Hospital and was unwell after returning home. She has nausea, vomiting and headache. She consulted her general practitioner (GP) Dr Partlin who diagnosed concussion. She returned to work the following week. 6. On examination he noted Ms Green was 171cm in height and weighed 120kg. Medical Assessor Cameron found no cognitive impairment and reported Ms Green scored 30/30 on Mini Mental State Examination. Medical Assessor Cameron found a full range of motion at both knees, clicks and crepitus at both knees but no instability. He found no neurological abnormalities in the lower extremities. No gait abnormality was detected. Circumferences of the lower extremities were 57cm above the right and left knee and 41cm below the right and left knee. 7. Medical Assessor Cameron reviewed the MRI of the knees dated 15 August 2023, the Ambulance Report, the discharge summary from Sutherland Hospital, the reports of Dr Seeto, the MRI of the brain dated 14 September 2023, and the report of Dr Partlin dated 1 September 2023 (presumably the referral by Dr Partlin to Dr Wardman dated 1 September 2023). 8. Medical Assessor Cameron concluded Ms Green did not sustain a traumatic brain injury but may have sustained a soft tissue injury to the head. 9. In relation to the knees he stated:
"It is plausible that there was a meniscal injury at the left knee in the subject motor vehicle crash, in view of forced contraction and compression of a degenerative meniscus. It is noted that the imaging study suggested a recent injury to the meniscus. There is evidence of pre-existing degenerative changes in both knees with osteoarthritis."
"The injury to the left knee is not a threshold injury because there had been an injury (that can be termed a "rupture") to the meniscus which is composed of cartilage".
"... History of events - Pt was driving at approx. 50kms/hr when the tipper pulled out of a driveway, the pt states she tried to swerve and brake to avoid the tipper however couldn't avoid the collision, nil airbag deployment, pt was wearing a seatbelt, no cabin intrusion, Pt states nil LOC/headstrike, pt states she was still in the car because she was shocked not due to entrapment or pain, pt able to ambulate from vehicle to stretcher. OE A - patent and maintaining own, B - spontaneous and unlaboured, speaking in full sentences, RR and Sats BTF, chest sound clear, equal rise and fall of chest, C - warm and well perfused, no active bleeding noted, pt co chest pain on left side described more as breast pain ?from seatbelt, ECG SR, HR and BP BTFs, D - GCS 15, alert and orientated, states some pain in left breast, tender on palpation, no swelling or bruising noted, pain in bilateral knees, more so in left, able to weight bare as normal, offered paracetamol and ibuprofen which the pt has politely declined at this point, nil c-spine tenderness/headache/nausea/vomiting/dizziness ...".[12]
Sister and Mum both thought she was a little bit weird
Nausea on the train on Tuesday - almost had to get off
Feels like her body is very sensitive - toes hurt, fingers hurt".
"MVA in mid-June. Since then worsening pain in neck with some throbbing of the left arm. Tender in the midline around C7 ?Disc herniation with radiculopathy. Also worsening pain in both knees.
History of arthroscopy/meniscectomy on right and ACL reconstruction on left. Right knee pain internally on weightbearing, left knee pain worse over the lateral knee joint ?LCL ligament damage."[15]
Was prone and they flexed her knee and manipulated it
Could barely walk and had to be on crutches for several days
O/E: Appears well and comfortable Overweight Gait - relatively normal and independent Left knee: Almost full extension, flexion to 80 degrees Medial/posterior tenderness but no joint line tenderness medial joint line No significant effusion."
Last 4 days L knee has been very sore laterally/posteriorly
No locking but very restricted movt".[22]
"knee settling, able to exended better and flex quad
Has only been slightly better today due to resting all week."
"1. Status post previous neo ACL without evidence of complication.
"There is a disc protrusion at C6/7 level contacting and slightly distorting the cord but there is no high grade neuroforaminal stenosis or central canal stenosis seen secondary to this."
History of arthroscopy/meniscectomy on right and ACL reconstruction on let. Right knee pain. Internally on weight bearing, left knee pain wore over the later knee joint? LCL ligament damage.
A 6.8 x 1.2cm (SI x trans x A-P) complex Baker's cyst has progressed demonstrating some superomedial decompression demonstrating loculation and septations within the posterior femoral fat pad medically deep to the sartorius distal MTJ.
Status post remote bone patellar tendon bone neo ACL with some mature scar remodelling along the line of tendon harvest and a little reactive bony overgrowth along the bone plaque harvest sites which is slightly progressed. Some stable susceptibility artefact along the femoral tunnel and tibial interference screw fixations. The neo ACL remains maturely ligamentised, and intact.
The previously documented peripheral vertical tear of the anterior horn of the lateral meniscus has healed. The associated anterior parameniscal cyst has slightly changed in configuration today measuring 1.2 x 0.6 x 1.1cm(SI x AP x trans) extending into the midline. No acute tear is identified. A little nonspecific but likely degenerative signal has developed at the free edge of the body.
Lateral compartment articular cartilage, subchondral bone, the lateral collateral ligament and visualised lateral capsule remain intact.
The medial meniscus also remains intact. Medial compartment articular cartilage, subchondral bone and the visualised MCL are stable in appearance. There is trace tibial collateral and bursal fluid.
The remaining components of the extensor mechanism are intact.
Close to the superior joint line, a full thickness chondral fissure has developed at the lateral patellar facet close to the median ridge contiguous laterally with a short segment of deep surface chondral delamination measuring up to 0.2cm associated with a small focus of subchondral bone marrow oedema signal. Adjacent new 20% oblique chondral fissures just causal to the full thickness chondral fissure have also developed.
Some surface chondral fissuring at the mid medial patellar facet adjacent to the median ridge is now evident. Trochlear articular cartilage is intact allowing for a little new surface chondral fissuring at the medial joint line medical trochlea superiorly. The anterior fat pads define normally. The proximal tibiofibular articulation is intact.
The visualised static and dynamic posterolateral and posteromedial corner stabilisers define normally. No neurovascular abnormality is appreciated. Intact visualised ITB.
Mild static lateral patellar subluxation is evident associated with an increased lateral patellar tilt. Negligible (1cm) tibial tuberosity lateralisation.
There is physiological joint fluid in the suprapatellar pouch demonstrating synovitis.
A 1.3 x 0.7 x 1.7cm (trans x AP x S-I) ovoid sclerotic ossicle demonstrating some heterogeneous intermediate T2 signal is evident anteriorly overlying the mid lateral trochlea along the lateral joint line (series 3 image 19 and series 6 and 7 images 27). There are also two adjacent corticated marrow signal intensity intra-articular bodies measuring 0.4cm and 0.7cm within the deep midline anterior joint recess deep to the ligamentum mucosum outlined by trace fluid. Mild further loculation in the posterior joint recess where there is a synovitis and two further corticated marrow signal bodies measuring 0.4cm and 0.5cm just posterior to caudal fibres of the PCL. Minimal popliteus sheath loculation. No Baker's cyst.
There is some diminution in the volume of the medial meniscus suggesting a prior meniscectomy relatively sparing the root ligament attachments. Superimposed degenerative fraying and signal within the diminutive posterior horn, heterogeneous degenerative intrasubstance signal in the diminutive body posteriorly and blunted free edge truncation and a little heterogeneous degenerative signal within the diminutive anterior horn are evident. There is also confluent degenerative signal at the posterior horn posterior root ligament. junction and some static extrusion of the body (0.2cm) suggesting a degree of static dysfunction. Enthesophytes at the anterior ligament attachment.
Grade 4 medial femorotibial compartment OA with denudation of the medial weight bearing MFC and MTP articular cartilage extending to the medial joint line. Erosion and remodelling of the exposed subchondral plates with associated subchondral bone marrow oedema signal more confluent at the exposed medial joint line MTP with patchy involvement at the exposed MFC. Bulbous medial joint line MFC and transversely oriented MFC notch osteophytes. Tiny medial tibial spine osteophyte formation is also noted. There is medial bowing of the MCL which is otherwise intact.
There is the suggestion of a partially healed horizontal intermediate T2 signal tear involving the anterior horn of the lateral meniscus medially measuring up to 0.7cm extending to the inferior articular surface 0.5cm from the free edge.
Lateral compartment articular cartilage is preserved. There are small lateral joint line and LFC notch osteophytes. The lateral collateral ligament is intact. Intact lateral capsule.
Some degenerative signal in the PCL particularly at and proximal to its genu. Some medial displacement of the ACL by LFC notch osteophyte formation. The ACL is otherwise unremarkable.
The extensor mechanism is intact allowing for mild tendinosis at tuberosity and insertional fibres of the patellar tendon.
There is a button osteophyte at the upper medial trochlea laterally extending close to the trochlear sulcus measuring up to 0.7 x 0.6cm (SI x trans) reconstituting in the surface chondral contour. There is also a tiny deep chondral blister caudally at the medial patellar facet adjacent to the median ridge measuring 0.2cm. Marginal trochlear osteophytes larger medially. Intact anterior fat pads and proximal tibiofibular articulation.
The visualised static and dynamic posterolateral and posteromedial corner stabilisers define normally allowing for a small pes anserine bursal effusion having a depth of 0.3cm.
Draped anteromedially about the proximal tibial epiphysis, there is a 3.9 x up to 2.1 x 0.5cm (trans x SI x A-P) multiloculated septated cystic appearing structure demonstrating some synovitis (see discussion below).
"There is mild cerebral hemispheric microvascular ischaemic change which is somewhat in excess of what would normally be expected. ...
There is a lesion of the superficial lobe of the left parotid gland...".[26]
?ACL ? lateral meniscus. One day ago felt pop in knee, gave way. Swelling and pain lateral/posterior. History of ACL reconstruction and degenerative OA.
A small possible haemarthrosis has developed loculated in the suprapatellar pouch demonstrating some synovitis. Mild extension of the lateral meniscosynovial recess.
The moderate complex Baker's cyst is stable, again demonstrating some superomedial loculation. Some posterior periscapsular oedema signal about the posterior oblique ligament is evident without clear periscapsular oedema signal associated with the Baker's cyst to suggest a leak/rupture.
Since the previous study, a full thickness radial tear at the junction of the posterior horn and posterior root ligament of the medical meniscus has developed (series 7, images 11-12 and series 9, image 22 also visible on series 10, image 23) demonstrating a 'ghost sign'. Associated static extrusion of the body by up to 0.25cm is evident.
Medial compartment articular cartilage, subchondral bone and the MCL remain intact.
There has been further healing of the peripheral vertical/longitudinal tear of the anterior horn of the lateral meniscus which is now almost indiscernible. It is again contiguous with an under surface partially healed horizontal tear of the anterior horn/anterior root ligament junction. The associated dorsal parameniscal cyst overlying the meniscocapsular junction extending to the midline has slightly change in configuration, today measuring 1.4 x 0.5cm (trans x SI x AP).
The lateral compartment articular cartilage, subchondral bone, the lateral collateral ligament and lateral capsule define normally.
The remote patellar tendon bone neo ACL remains intact with a stable appearance at the graft harvest site. Uncomplicated tunnel. Clear anterior intercondylar notch. Intact PCL.
The remaining components of the extensor mechanism define normally.
The previously documented features of Grade II chondromalacia patellae are unchanged allowing for slightly reduced bone marrow oedema signal in association with a full thickness chondral fissure at the lateral patellar facet close to the superior joint line adjacent to the median ridge.
The visualised static and dynamic posterolateral and posteromedial stabilisers also remain intact. No neurovascular abnormality has developed.
"MRI of the left knee on 15 August 2023 is reported as showing a Baker's cyst and intact bone patellar tendon knee OA ACL, an interval healing of a peripheral vertical tear of the anterior horn of the medial meniscus with some change in the configuration of the associated anterior parameniscal cyst, features of progressed chondromalacia".
"It is plausible that there was a meniscal injury at the left knee in the subject motor vehicle crash, in view of forced contraction and compression of a degenerative meniscus. It is noted that the imaging study suggested a recent injury to the meniscus. There is evidence of pre-existing degenerative changes in both knees with osteoarthritis."
"? ACL? Lateral meniscus. One day ago felt pop knee, gave way. Swelling and pain lateral/posterior. History of ACL reconstruction and degenerative OA".
Pre-accident medical history and relevant personal details
T-boned the left front corner of her vehicle. She had a seatbelt on. The airbags did not deploy. 2. The Medical Assessors asked Ms Green how she hurt her knees, and she said that she felt that her knees moved forward and over the top of her shins at the time of the impact, but she didn't have any actual impact of the knees on the dashboard or other object. 3. Ms Green had sore knees after the accident, especially the left knee. She doesn't recall any bruising to the knees, although she had bruising to the chest from the seatbelt. She recalls having some sort of shock reaction and she "just froze" and sat in her car. Bystanders assisted her out of the car. 4. The ambulance arrived and the paramedics assessed her, and she was found able to walk to the ambulance. Ms Green said she does not recall the paramedics palpating her knees, but she did report pain in the knees and chest. Police and fire brigade were also on scene.
History of symptoms and treatment following the accident
Details of any relevant injuries or conditions sustained since the accident
[5] Briggs v IAG Limited Trading as NRMA Insurance [2022] NSWSC 372.
Youi Pty Limited
Green [2025] NSWPICMP 425