austlii:nsw/NSWPICMP/2025/317
General information, not legal advice.
NSWPICMP
2025-05-07
Original judgment source is linked above.
"One may accept that a review panel is engaged in a process of dispute resolution by expert assessment of medical issues arising under the Act. However, the questions arise in a legal context and it is incumbent upon the panel, medical practitioners they may be, to correctly apply the law including the law of causation in the exercise of their powers. This includes the provisions of Division 3 of Part 1A of the Civil Liability Act 2002 (NSW), ss5D and 5E: see s 3B(2)(a) of that Act. Although it may be expected that questions about the appropriate scope of liability will arise but rarely."
"There is no simple common test of causation that is applicable in all cases, but the accepted approach involves determining whether the injury (and the associated impairment) was caused or materially contributed to by the motor accident. The motor accident does not have to be the sole cause as long as it is a contributing cause, which is more than negligible."
25 August 2023.[31] Dr Rastogi opined that the motor accident caused post-traumatic stress disorder with anxiety. 2. Dr James Bodel, orthopaedic surgeon, was qualified by the claimant and provided a report dated 7 September 2024.[32] The doctor noted that the X-rays taken at hospital showed no fractures. 3. Dr Bodel opined that the claimant suffered a soft tissue injury to the neck, rotator cuff injury to the left shoulder, soft tissue injury to lower back as well psychological sequelae. The doctor assessed whole person impairment of left upper extremity at 8%, cervical spine at 5% and the lumbar spine at 5%. 4. We note that the opinion of Dr Robin Mitchell, referenced in submissions, was not in the bundles provided to the Panel.
Insurer's letter dated 13 July 2023[33]
Insurer's submissions dated 16 January 2025[34]
"Mrs Tabassum attended the medical suites at PIC on 23 April 2025. She was unaccompanied.
Mrs Tabassum stated that she was in good health prior to the accident and had been working full-time as a dental assistant. She is married and lives with her husband and now has a one-year-old daughter. She migrated from Bangladesh in 2018. There were no previous injuries to those assessed today. She has recently completed a Masters in Public Health.
Mrs Tabassum was a pedestrian and hit by a car at speed. She stated that the collision was on her left side of the body causing her to fall onto the right side on the road. The ambulance and police attended the scene of the accident, and she was taken to Liverpool Hospital for assessment where x-rays and CT scans were done.
History of symptoms and treatment since the accident
At Liverpool Hospital, abrasions were noted to the left lateral thigh, and left elbow but x-rays reported no fractures of the left hip, femur and left shoulder. After a one-day admission she was discharged and followed up by her treating GP 2 days later, who prescribed analgesics and recommended one month of rest. She was later referred to physiotherapy and rehab.
Mrs Tabassum returned to work but attended the hospital again with vomiting and faintness. She also transferred GP as their address relocated. There were follow-up consultations with a psychologist.
The physiotherapist treated her left shoulder, left upper leg and low back. The treating GP referred her to a neurosurgeon, Dr Abrazho who organised an MRI, and ultrasound of the left hand and nerve conduction studies of the upper limbs by Dr Griffith. She consulted a pain specialist Dr Richmond who noted cervical spine and left shoulder pain radiating into the left hand and low back pain with pain in the left buttock. Nerve conduction studies of the left median and radial nerves were normal. Dr Richmond has been treating her in a pain management group in 2024.
More recently, Mrs Tabassum has consulted a neurologist, Dr Abdul Manun on 31 March 2025 who requested a left occipital nerve block and then the right occipital nerve block which has not been approved for treatment yet.
Mrs Tabassum states that there had been no further injuries sustained since the accident.
Mrs Tabassum has left-sided neck pain but occasionally on the right which is variable in intensity she states that the left hand gets cold and stiff occasionally with discomfort in the left shoulder, but the right arm and shoulder are asymptomatic. There is lumbar pain centrally and laterally in the left which increases with bending or carrying her daughter. She gets relief from wearing a lumbar brace whenever she is mobile.
There is pain and numbness in the left leg but the right leg is asymptomatic.
Mrs Tabassum is a non-driver and walks 10 minutes at a time. She continues to work as a dental assistant for 8 hours 4 times a week and catches the train and bus to work.
Present medication is gabapentin one at night, Celebrex 200 mg a day, amitriptyline 50 mg at night, Panadeine Forte one at night, Panadol 2 tabs 3 times a day and medication for hypertension.
She states that physiotherapy was helping but funding ceased 2 months ago. She consults her GP when necessary and has a consultation arranged with her new neurologist Dr Manum in July for follow-up and possibly Dr Richmond, the pain specialist.
Mrs Tabassum walked into the rooms with a normal gait and sat comfortably during the interview. She stated she is right-handed. The height was measured at 158 cm and weight of 78 kg. Mrs Tabassum was wearing a lumbar brace which she says has been utilised long-term.
On testing range of movement, flexion/ extension to 50% of expected range and side bending and rotation were 60% of expected range bilaterally with no asymmetry. On palpation there was tenderness bilaterally in the suboccipital region and she is very sensitive to touch in the left trapezius muscle and lateral left neck, but no guarding or spasm was noted.
On neurological examination of the upper limbs, reflexes were equal and brisk bilaterally with normal power and a global decrease in sensation in the left arm to light touch which was not in a dermatomal pattern. No muscle wasting was apparent with circumferences the upper arms 28 cm bilaterally (10 cm above the olecranon process) and in the upper forearm 24 cm bilaterally (5 cm below the olecranon process).
There was a normal range of movement of the elbows, wrist and hands with no muscle wasting noted on examination on testing at the hands.
Mrs Tabassum walked with a normal gait but was unable to walk on heels and toes due to unsteadiness. On testing range of movement, flexion/extension was 50% of expected range and side bending was 70% of expected range bilaterally with no asymmetry. On palpation there was tenderness globally over the lumbar musculature, but no guarding or spasm was noted. Straight leg raise when lying was 80° on the right and 60° on the left which was limited by pain in the left thigh and calf. Sciatic nerve root tension signs were negative. There was a full range of movement of the knees.
On neurological examination of the lower limbs, reflexes were equal bilaterally with normal power and no sensory changes were noted. No muscle wasting was apparent with the circumferences of the lower thighs 44 cm bilaterally (10 cm above the superior patella pole) and at the maximum circumference of the calves 36 cm bilaterally.
On inspection of the shoulders, no muscle wasting was apparent and on passive movement there was a normal range of movement of the right shoulder but pain on any passive movement greater than 90° abduction of the left. On palpation there was a global complaint of pain to light touch of the left shoulder with no referral of pain from the cervical spine. Active movements were measured using a goniometer and repeated 3 times.
I noted that there had been a significant decrease in range of movement of the left shoulder in the last 6 months. Mrs Tabassum stated that in this time physiotherapy had been ceased which increased pain in the left shoulder region. I explained to her that due to this recent deterioration in range of movement it was not possible to determine impairment by this method and another method would be needed to be used. She stated that she understood this reasoning."
Mrs Tabassum stated that her left hand and arm gets cold at times but on examination no abnormality was noted, and nerve conduction studies were negative. There was no evidence of a carpal tunnel syndrome. Considering the absence of pathology in the left hand, the absence of explanation of how the left hand was injured in the motor accident and the clinical examination by Medical Assessor Moloney, there is no assessable impairment of the left hand. 11. Mrs Tabassum complained to her GP two days after the accident that she had left shoulder pain which was recorded in the certificate of capacity. The Panel accepts that Mrs Tabassum sustained a soft tissue injury to the left shoulder in the subject accident when she was struck by the vehicle and fell to the ground. This condition was also treated by a physiotherapist. 12. There has been a significant decrease in range of movement this year in comparison to the previous documentation by Medical Assessor McGrath. Mrs Tabassum states that due to increased pain in the last six months she has had significant loss of range of movement and attributed this to cessation of physiotherapy. 13. Medical Assessor McGrath assessed loss of range at 1% impairment of the left shoulder. In October 2023 the physiotherapist recorded "shoulder active range is full with pain at end range".[39] Medical Assessor Moloney on behalf of the Panel found an 8% impairment due to loss of active movement at the recent examination. 14. In these circumstances, the gross change in loss of motion over the last six months is not medically explicable despite the cessation of physiotherapy. There is inconsistency of the left shoulder movement as found by Medical Assessor Moloney within the meaning of cl 6.41 of the Guidelines. 15. The Panel is required to modify the impairment estimate in accordance with cl 6.40 of the Guidelines in light of this finding of inconsistency. The left shoulder is assessed by analogy by using the gleno-humeral (shoulder) joint in Table 18 of AMA 4 which is 36% permanent impairment for complete loss. Applying Table 19 of AMA 4 this is mild crepitus which is 10% of joint impairment. Accordingly, providing a beneficial analogy in favour of the claimant, we assess left shoulder impairment at 3.6% rounded up to 4% permanent impairment. 16. There are no relevant pre-existing physical symptoms and no objective evidence of impairment at the time of the motor accident. There is no basis to make any deduction for pre-existing impairment. 17. We are satisfied that the impairment is permanent within the meaning of cls 6.19 and 6.20 of the Guidelines because the condition is well stabilised, the claimant does not require further surgery and treatment in the foreseeable future. The suggestion of proposed treatment by Dr Manun will have no impact on potential assessable impairment as it is designed to relieve occipital pain. 18. The Medical Assessor's clinical examination of the claimant and the Panel's view is that there is unlikely to be a change of greater than 3% impairment over the next year.
[2] See Division 7.5 and Schedule 2, cl 2 of the MAI Act.
[5] Section 7.26(5) of the MAI Act; claimant's bundle, page 303.
[8] See s 3B(2) of the Civil Liability Act 2002.
[9] [2021] NSWSC 13 (Raina) at [65].
[37] [2021] NSWCA 287 at [40], [41] and [45].
[38] [2022] NSWCA 31 at [11], [21], [64].
Tabassum
Allianz Australia Insurance Limited [2025] NSWPICMP 317