austlii:sa/SAET/2025/83
General information, not legal advice.
SAET
2025-08-12
Original judgment source is linked above.
(2) Subsection (1) operates -
(a) subject to the qualification that if a worker suffers an injury of a kind referred to in the first column of Schedule 2 and has been employed in work of a type referred to in the second column of Schedule 2 opposite the injury, the worker's injury is presumed, in the absence of proof to the contrary, to have arisen from employment; and
(b) subject to Schedule 3.
(3) If a worker retires or is retired from employment on account of age or ill‑health and the worker makes a claim for noise induced hearing loss after the expiration of 2 years from the date of the retirement, subsection (2)(a) does not apply in relation to that claim.
Most of the time it was reasonably quiet, depending, in the actual building itself. No, not up in the plant rooms or when you went up on the roof to blow pine needles off the roof or stuff like that.
Constant and prolonged noise in the workplace where noise levels were unacceptable.[1]
Depending on the area I was working in, like plant rooms and stuff like that. They were constant and very noisy.
In the claimant's statement dated 27 March 2024, he detailed the duration and frequency of use which was clearly much greater than what has been observed and assessed by me and detailed by other maintenance personnel at different aged care sites.[4]
Mr Doyle: Looking, sort of, empirically, in the circumstances we've just discussed, it's not surprising, is it, that if we look at a cross-section of the community, it's a common experience that as people age many of them suffer an increase in - well, a deteriorating hearing?
Dr Raj: That's correct. So as we age - I mean, the deterioration of hearing depends on several causes. I mean, there's a natural cochlear cell degeneration that occurs, like any central brain cell. So as we get older, the brain cells, the number of brain cells we get, get reduced, or function less. So that sort of thing happens in the cochlear as well. So that that's one, and then you may have other effects. For example, if you have hypertension, diabetes, autoimmune disease, rheumatoid arthritis, and if your small vessels are affected, it can affect at the same time. So they can all occur at the same time. But how much of it - how much of each of the conditions contribute, you cannot say or exactly determine.
So there are studies that indicate that - I'm not giving you the exact figures, but these are approximate figures because I have come to know about them from a very long time ago, but there are studies indicate that for - if every - of 100 people who are exposed to noise, only 20 to 30 of them will actually suffer noise injury, and this is in relation to the cochlear sensitivity, because not all cochleas are sensitive to noise, and there are other variables as well that can affect the noise injury. So the studies indicate that if 100 people are exposed to noise, then about 30, an average of 30 per cent would get noise injury, and the other 70 per cent would have a hearing loss, they might have been exposed to noise but may not have a noise injury.
Mr Doyle: So is it fair to say, Doctor, that noise-induced hearing loss is typically associated with continuous exposure at certain levels? I mean continuous in the sense that whether it's four hours a day continuous, or eight hours a day continuous, or six hours a day continuous, that's the typical scenario. The continuity of exposure is the typical - sorry - and the decibel level, is the typical feature of noise-induced hearing loss.
Dr Raj: That's correct. So what we're trying to establish here is that at a particular noise level, he's been exposed to a particular duration during the day, and that continues every day, and because it continues every day, and the cochlear needs more time to recover than what is given, therefore, it is a continuous damage or continuous exposure that occurs. Yes. That's exactly what we mean.
Mr Doyle ...your basic assumption in Mr Ludlow's case was that, essentially, day in, day out, when he was at work, he was being exposed to noise levels which one might say were at least the level of four hours at 90 decibels or two hours at 100. Sorry, I got that wrong. Yes. Four hours at 90 or two hours at 100. That sort of level.
Dr Raj: Yes. From the history he gave me. Yes...
Mr Doyle: ...in other words, those high levels of exposure, perhaps at 90 or 100 decibels for a very short period of time occurred only intermittently, for example, every month or a couple of months or every few weeks; that is, once every few weeks. That level of exposure wouldn't be expected to cause noise-induced hearing loss?
Dr Raj: Yes. Correct. I agree with that.
Mr Doyle: But in any event, if we go back to your underlying assumption in Mr Ludlow's case, that whether the exposure was two hours a day or four hours a day or more at levels of 85 or 90 decibels or even 100 decibels, unless that exposure was on a continuous basis, that is day after day and week after week, that it's not exposure which would be likely to cause noise-induced hearing loss.
Dr Raj: So let me try to get this correct, what you just said. So we are assuming that he - sorry, can you repeat the whole thing, so I can
Mr Doyle: What I'm saying is if those levels of exposure were only truly intermittent; in other words, those high levels of exposure, perhaps at 90 or 100 decibels for a very short period of time occurred only intermittently, for example, every month or a couple of months or every few weeks; that is, once every few weeks. That level of exposure wouldn't be expected to cause noise-induced hearing loss?
Dr Raj: Yes. Correct. I agree with that.
...the loss is not linear. So when you would draw the graph of how the noise-induced hearing loss occurs, in the first five, six, seven, 10 years, 15 years of noise exposure, the cochlear injury is significantly more than the next 15 years of noise exposure.
The work history and noise survey do not suggest noise exposure during this employment sufficient to have damaged the hearing. Exposure to recreational noise may have contributed.[8]
Although the pattern of the latest audiogram is consistent with noise induce hearing loss, causes other than noise exposure can produce a similar pattern.[9]
The increase in the high tone hearing loss is 8.8% and it seems unlikely that this can be explained by 9 years of exposure to intermittent noise...
The conclusion must be that there is another cause or causes of the hearing deterioration the nature of which is uncertain.[12]
It is axiomatic that noise induced hearing loss does not progress after leaving the noise that caused it. The audiograms dated 22 September 2021 and my own audiogram show significant deterioration in the frequency usually measured for noise induced hearing loss Using 2000-4000 cps inclusive and adjusting for his age the hearing losses are 10% and my own result is 12.4%.
Although both the more recent audiograms have a pattern consistent with noise damage, such a pattern is by no means diagnostic and occurs with many degenerative inner ear conditions that are not work related.[13]
There are major problems with the conclusions reached by Dr Raj.
Noise induced hearing loss progresses very slowly and more slowly later in a noise career. Typically loss occurs mostly in the first decade of exposure to noise capable of causing it and therefore the rate of deterioration slows dramatically. Progression from 2.6% on the pre-employment audiogram to 11.4% (the average of the later audiograms) is highly unlikely to be explicable in terms of noise exposure.
Intermittent noise exposure as described by Mr Ludlow at Uniting SA is much more likely to damage the hearing than continuous noise because the ears have time to recover between episodes of noise exposure.
The Dewing noise survey based on analysis of several care organisations demonstrates that maintenance workers employed are unlikely to be exposed to noise sufficient to damage the hearing. Mr Dewing does concede that on a particularly busy day with special projects, it is possible that the noise exposure exceeded the safe levels on those days.
Mr Dewing found that the duties claimed by Mr Ludlow as a maintenance worker were well in excess of those he has encountered in similar care homes. He formed the opinion that Mr Ludlow was exaggerating his noise exposure.
I agree that the recreational noise described is unlikely to have contributed to the hearing loss.
Conclusion
Mr Ludlow has progressive hearing loss that has increased from 2.6% , shortly after he joined Uniting SA, to 11.4% currently and for reasons stated this cannot be explained by intermittent noise exposure for 3 years during this employment.
The hearing loss is more likely to be degenerative in type, in excess of that expected from ageing. The exact nature of the degeneration is uncertain and unlikely to be elucidated by further investigation.[14]
Industrial noise deafness progresses very slowly and more slowly later in a working career. It is often described as a decelerating injury. 7.4% in 7 years (the loss in 2021 minus that in 2014) is most atypical for intermittent noise exposure, particularly as in this case Mr Ludlow was exposed to intermittent noise for only 7 years before he became a supervisor and less directly exposed to the noise of power tools.
I have reviewed the video link of a Go-Kart engine revving. Intermittent noise of this nature is unlikely to be a problem since it is the average noise over an 8-hour period that determines the risk to hearing.
I remain of the opinion that Mr Ludlow's hearing loss progression after 2014 is unlikely to be noise induced for the reasons explained.[17]
No, I think the main reason was I had had the opportunity to revise my previous report, and I think at that point I realised that there was something wrong here, that an increase in the hearing loss of that degree, when there had only been some two per cent after 20 years as a toolmaker, just didn't add up. So this was an opportunity, really, to revise my opinion.
Experts do not come to the courtroom as disinterested observers. They are generally sought out by one side or other side in legal proceedings, requested to make a report to legal representatives, paid for that and, if the report coincides with the arguments which the lawyers wish to put forward in the case, asked to testify. Experts are no more altruistic than lawyers. They expect remuneration and they receive it. Of necessity, this affects the relationship between the expert and the side for which he or she appears. There are all manner of pressures, albeit often subtle ones, that propel the expert in the direction of making findings which are acceptable to the side for which he or she initially prepares the report and then is asked to appear. Some of the pressures are financial but just as important are those which may be summed up as 'cause identification': [19]
...[the] discharge of the burden of proof depends on experience by the trial judge of an actual persuasion of the existence of the matter to be proved. There must be an appropriate degree of confidence in its existence."[20] (Emphasis added mine)
...the judge is not bound always to make a finding one way or the other with regard to the facts averred by the parties. He has open to him the third alternative of saying that the party on whom the burden of proof lies in relation to any averment made by him has failed to discharge that burden. No judge likes to decide cases on burden of proof if he can legitimately avoid having to do so. There are cases, however, in which, owing to the unsatisfactory state of the evidence or otherwise, deciding on the burden of proof is the only just course for him to take.[22] (Emphasis added mine)
1 The application for review is dismissed.
2 I grant the parties liberty to apply as to costs.
[18] (1999) 76 SASR 95 at 100.
[19] Freckelton; The Trial of the Expert, Oxford University Press (1987) at 124.
[20] [2000] NSWCA 255 at [61] per Heydon JA.
Ludlow
Return to Work Corporation of South Australia [2025] SAET 83
(1999) 76 SASR 95