austlii:nsw/NSWPICMP/2025/265
General information, not legal advice.
NSWPICMP
2025-04-16
Original judgment source is linked above.
Kelly v Insurance Australia Limited t/as NRMA Insurance; Kelly v RACQ Insurance Ltd [2025] NSWPICMP 265
MOTOR ACCIDENTS - ; review of Medical Assessment Certificate (MAC); assessment of threshold injury and permanent impairment disputes; first insurer (NRMA) denied statutory benefits beyond 26 weeks on basis that all of claimant's injuries are threshold injuries; second insurer (RACQ) admitted liability for the damages claim but did not concede that the claimant's accident-related impairment exceeds the 10% threshold; Medical Assessor (MA) did not assess whole person impairment (WPI); found that all referred injuries were threshold or not caused by the subject accident; Review Panel found two signs of radiculopathy in lumbar spine and lower limbs; - MAC revoked and new certificate issued; assessed 11% WPI for lumbar spine and right ulnar neuritis.
Certificate issued under s 7.23(1) of the Motor Accident Injuries Act 2017 (the Act)
(a) The following injury caused by the motor accident:
(i) shoulder - right shoulder soft tissue injury,
is a THRESHOLD INJURY for the purposes of the Act.
(b) The following injuries caused by the motor accident:
(i) lumbar spine - L5 protruded disc with pain daily lumbosacral disc lesion and left leg radiculopathy;
(ii) arm - right ulnar nerve neuritis;
(iii) leg - pain radiating all the way down at the left leg to the foot, numbness and tingling in the left foot, and
(iv) leg - sciatic pain mostly left, occasionally on right,
is not a THRESHOLD INJURY for the purposes of the Act.
(c) The following injuries referred to the Review Panel for assessment have been assessed and determined to be not caused by the motor accident:
(i) colorectal - intermittent bowl disturbance, and
(ii) knee - bilateral knees pain.
A decision as to whether these injuries are a threshold injury is not required for the purposes of the Act.
REVIEW PANEL ASSESSMENT OF PERMANENT IMPAIRMENT
Certificate issued under s 7.26(7) of the Act
(a) The following injuries caused by the motor accident give rise to a permanent impairment of 11% and is GREATER THAN 10%:
(i) lumbar spine, and
(ii) right ulnar neuritis.
The following injury caused by the motor accident:
is a THRESHOLD INJURY for the purposes of the Act. An assessment of the degree of permanent impairment of these injuries is therefore not required.
The following injuries referred to me for assessment have been assessed and determined to be not caused by the motor accident:
A decision as to whether these injuries are a threshold injury is not required for the purposes of the Act.
The following injuries referred to me for assessment have been assessed and determined to be not caused by the motor accident:
A decision as to whether these injuries are a threshold injury is not required for the purposes of the Act.
The following injury caused by the motor accident:
is a THRESHOLD INJURY for the purposes of the Act. An assessment of the degree of permanent impairment of these injuries is therefore not required.
The following injuries caused by the motor accident give rise to a permanent impairment which IS NOT GREATER THAN 10%:
Of the injuries referred for assessment nil injuries are related to the motor accident and therefore the permanent impairment IS NOT GREATER THAN 10%:
(a) radiculopathy was present;
(b) causation, and
(c) failure to assess WPI.
"Radiculopathy was present with respect to the lumbar spine
The first insurer submits the clinical examination findings of the Medical Assessor do not show radiculopathy in line with the Motor Accident Guidelines (Guidelines).
The first insurer submits the Medical Assessor diagnosed a soft tissue injury to the lumbar spine as a result of the subject accident.
The first insurer further submits the Medical Assessor clearly stated that radiculopathy, as per the Guidelines, is not currently present, nor has it been present since the accident.
The first insurer submits there is no medical evidence to establish the presence of radiculopathy, as a result of the subject accident, and the Medical Assessor has not erred in his determination.
The first insurer submits the Medical Assessor clearly advised that the injuries described in relation to the right and left legs were actually symptoms arising from the lumbar spine and not the legs themselves.
The first insurer submits there were no clinical findings on examination of the lower limbs that could be ascribe to a separate injury of the legs.
The first insurer submits the Medical Assessor correctly attributed the reported symptoms to the lumbar spine in his assessment.
The first insurer submits the WPI assessment (for which the second insurer is on risk) has no bearing on the threshold injury dispute."
"6.5 An assessment of the degree of permanent impairment is a medical assessment matter under cl 2(a) of Schedule 2 of the Act. The assessment must determine the degree of permanent impairment of the injured person as a result of the injury caused by the motor accident. A determination as to whether the injured person's impairment is related to the accident in question is therefore implied in all such assessments. Medical Assessors must be aware of the relevant provisions of the AMA 4 Guides, as well as the common law principles that would be applied by a court (or the Personal Injury Commission) in considering such issues.
6.6 Causation is defined in the Glossary at page 316 of the AMA 4 Guides as follows:
Causation means that a physical, chemical or biological factor contributed to the occurrence of a medical condition. To decide that a factor alleged to have caused or contributed to the occurrence or worsening of a medical condition has, in fact, done so, it is necessary to verify both of the following:
This, therefore, involves a medical decision and non-medical informed judgment.
6.7 There is no simple common test of causation that is applicable to all cases, but the accepted approach involves determining whether the injury (and the associated impairment) was caused or materially contributed to by the motor accident. The motor accident does not have to be a sole cause as long as it is a contributing cause, which is more than negligible. Considering the question 'Would this injury (or impairment) have occurred if not for the accident?' may be useful in some cases, although this is not a definitive test and may be inapplicable in circumstances where there are multiple contributing causes."
"The question of causation of injuries was not dealt with in Part 5 of the Guidelines but causation was addressed in Part 6, which related to assessment of permanent impairment. There is no reason to think that different principes were intended to be applied when a medical assessment was being made in relation to causation of minor injuries."
"Consider whether the motor accident did cause or contribute to (the claimant's condition). This require, not a consideration of material derived as a result of an internet search... but rather a consideration of the material referred to in 5.6 of the Guidelines, namely all the evidence available to the Panel, including all relevant findings derived from:
(1) a comprehensive, accurate history, including pre-accident history and pre-existing conditions;
(2) a review of all relevant records available at the assessment;
(3) a comprehensive description of the injured person's current symptoms;
(5) diagnostic tests available at the assessment, noting that imaging findings that are used to support the assessment should correspond with symptoms and findings on examination."
"5.3 The assessment will determine whether the injury related to the claim is a soft-tissue injury or a threshold psychological or psychiatric injury caused by the motor accident.
5.4 Diagnostic imaging is not considered necessary to assess threshold injury.
5.5 A diagnosis for the purpose of a threshold injury decision must be based on a clinical assessment by a medical practitioner or other suitably qualified person independent from the insurer.
5.6 The assessment of whether an injury caused by the accident is a threshold injury for the purposes of the Act should be based on the evidence available and include all relevant findings derived from:
Imaging findings that are used to support the assessment should correspond with symptoms and findings on examination."
Report by Dr T Mastroianni, consultant occupational physician, to the claimant's lawyers
No X-rays were reviewed. The following MRI report was on file:
Disc degenerative change and small disc protrusion from L3 to the sacrum. At L5/S1 right paracentral disc protrusion abuts but does not compress or displace the traversing right S1 nerve root. Probable degenerative changes at the right S1 joint.
As a result of the subject accident, the claimant sustained a soft tissue injury to the cervical spine from which he has recovered. He also injured the right arm and has ulnar nerve neuritis. In the lumbar spine, he aggravated pre-existing asymptomatic lumbar spondylosis and sustained a lumbosacral disc lesion. He has back pain and left leg sciatica and radiculopathy. My clinical diagnosis is:
The claimant has suffered a non-minor, non-threshold injury. He has sustained nerve injury causing radiculopathy and has also sustained a disc protrusion in the lumbar spine, being a complete or partial rupture of the fibrocartilaginous tissue and he therefore makes the criteria of a non-threshold injury as a result of both conditions. The injury to back and arm are the result of the subject accident. Prognosis is guarded.
The claimant has a lumbosacral disc lesion with left leg radiculopathy. He falls into DRE III (AMA 4, page 110, Table 2). This equates to 10% WPI.
He has right ulnar nerve neuritis and I assess grade 4 sensory deposit, 25%. The ulnar nerve equates to 7% upper extremity impairment for sensory deficit (AMA 4, Table 15). 25% of 7% is 1.7% which equates to 2% upper extremity impairment, which equates to 1% WPI.
The combined total is 11% WPI as a result of injuries sustained in the subject accident.
Report of Dr James Bodel, orthopaedic surgeon, to the claimant's lawyers
There were no X-rays or other tests available for review today. I have carefully perused the local doctors continuation notes which are consistent with the ongoing medical management of this gentleman's injury. He has significant disc pathology principally at the L5/S1 level with compression of the S1 and L5 nerve roots to the right hand side. His clinical symptoms today appeared to be mainly L5 and not S1. The knee and ankle reflexes are diminished slightly.
The local doctors continuation notes from the time of the accident confirm that he presented after the accident during work. There is an extensive treatment from the local doctor at that time..... the doctor does report that there was a previous back injury 20 years earlier with left sided sciatica which settled within a few weeks. I was not given that history but that is not unreasonable.
Following the injury, he had various medications. He later did have the further tests including the MRI scan showing the disc pathology and he was then referred to see the physios only. He has never been referred to a neurosurgeon or to any other specialist for review in spite of the history of numbness over the dorsum of the foot and possible cauda equina symptoms. He does not have any hyper-reflexia or other abnormality neuron signs that I can identify here today but he really should be assessed by a neurologist or medico-legal purposes and by a neurosurgeon from a treatment point of view.
The injury to the back was caused by the motor vehicle accident. He has some pre-existing degenerative change but he was asymptomatic and working without difficulty until that event. The body part involved is the lower part of the back with left-sided sciatica.
Dr Bodel says that the claimant has been left with a permanent impairment but is whole person impairment assessment is not in evidence.
Report by Dr Amanda Woodward for MRI lumbosacral spine
Vertebral body alignment is normal. No evidence of congenital canal stenosis or boney anomaly. Distal spinal cord is unremarkable with the tip of the conus at mid L1.
Disc height, hydration and contour are within normal limits from T12 to L3 and the intervening nerve roots exit normally.
L3/L4: mild loss of disc height, disc desiccation and mild broad based disc protrusion. No evidence of focal lesion of concern. Exiting and traversing nerve roots are normal.
L4/L5; disc desiccation, mild broad based disc protrusion. No significant canal stenosis, existing and traversing nerve roots are normal.
L5/S1: disc desiccation, mild loss of posterior disc height, small right paracentral disc protrusion. This abuts but does not compress or displace within traversing right S1 nerve roots. L5 nerve roots exit normally.
Throughout the lumbar spine, there is no evidence of acute boney injury or paravertebral soft tissue abnormality.
There is a small focus of subchondral bone marrow oedema in the right margin of the sacrum at the sacroiliac joint at the S2 level. This may reflect some degenerative change. No definite evidence of fracture. Left S1 joint is unremarkable. Elsewhere sacral architecture is normal without evidence of boney or soft tissue abnormality. Pre-sacral soft tissues are normal.
Disc degenerative change and small disc protrusions from L3 to the sacrum. At L5/S1 right paracentral disc protrusion shuts but does not compress or displace the traversing right S1 nerve root. Probable degenerative change at the right S1 joint.
The spine has been imaged from the inferior end plate of T11 to S2. There are five conventional lumbar-type vertebral bodies. The vertebral body heights are preserved. No fracture identified. No osseous lesion. There is Modic type 2 changes noted along several end plates.
There is multi-level disc height loss with reduced signal intensity in keeping with the degenerative disc disease and disc desiccation. There are end plate osteophytes seen at each of the lumbar levels most pronounced at L2/L3 and L3/L4.
No pre-vertebral soft tissue swelling. The posterior paraspinal musculature outlines normally.
The conus medullaris terminates at L1. The partially imaged cord is normal in size and signal intensity. There is normal configuration of the cauda equina nerve roots.
T12/L1: no disc bulge or focal herniation. Mild right facet degenerative changes.
L1/L2: mild loss of disc height with mild annular bulging. No significant canal for foraminal stenosis. Mild facet degenerative changes.
L2/L3: There is mild disc height and generalised disc osteophyte complex which indents the anterior thecal sac and narrow scalateral recesses. It appears to contact the descending L3 nerve roots without evidence of displacement or impingement. There is mild bilateral foraminal stenosis. The light lateral portion of the disc osteophyte complex also appears to contact but not displace or compress the (report end).
R2 Second insurer's submissions dated 10 October 2024 in reply to review application.
Failure to conduct assessment pursuant to the Guidelines
Failure to take into account/consider relevant considerations
Failure in determining the left and right leg injuries were symptoms, not injuries
The second insurer finally submits that there is no error in the certificate and no reasonable cause to suspect there are any material errors in the certificate as alleged by the claimant.
Date of Examination: 14/2/2025 at PIC Medical Suites, Sydney
Pre-accident medical history and relevant personal details
He said he now lives alone in his house in Tamworth. At the time of the motor vehicle accident, his father lived with him in the same house. He has since passed away.
At the time of the accident, he worked as a chef and courier driver but in late 2018 he gave up chef work because of needing to look after his family members in the evenings.
Regarding past back complaints, he said in 2012 he picked up his 350kg Harley Davidson motorcycle which had fallen on its side, and developed low back pain and sciatica in the left leg as far as the calf. He had two or three sessions of physiotherapy and thereafter did home exercises. There was no numbness in the leg. He was working as a hotel chef at the time but doesn't recall having any lost time from work. This episode settled.
He has had gout in the past affecting the left hallux and is treated with Allopurinol. He has a Baker's cyst at the right knee which seemed to resolve itself.
He has a history of GORD (gastro-oesophageal reflux disease) and has taken Somac for some years.
He drinks alcohol on the weekends and smokes roll-your-own cigarettes.
He doesn't recall any previous problems to the neck, right shoulder or right wrist.
Mr Kelly said on 8/5/2019, he was driving his own utility vehicle with no passengers. He was driving to a depot at Quirindi to pick up the courier vehicle. A car came through a Give Way sign on his left and hit the left side of his vehicle in a T-bone fashion. It destroyed the left front mag wheel on his vehicle and stoved in the left front panel.
The airbag deployed and hit him in the face and forced his head back. His car was written off. He was wearing a seatbelt at the time. He was holding the steering wheel and at the time of the impact the wheel moved forcefully to the left, wrenching his right hand and forearm around to the left.
Ambulance and Police Officers attended. He was assessed by the paramedics. He was complaining of some soreness in the right arm and neck and told to follow-up with his medical practitioner as required.
History of symptoms and treatment following the motor accident
He developed low back pain a couple of days after the accident. He noticed this after he had got up to walk to work at the local depot on 11/5/2019, and had central stabbing low back pain but no leg pain at that time. After he had arrived at work, his foreman drove him to Tamworth Base Hospital. The low back pain was excruciating that day. He even had difficulty lying on the plinth to have a CT scan done. He was certified unfit for work and given Oxycontin and referred back to his GP for follow-up.
He saw the GP on 16/5/2019 for the first time since the accident, after the visit to Tamworth Base Hospital.
A MRI scan which was done on 3/6/2019 because of complaints of left leg pain, low back pain, and numbness in the lateral left leg and to the dorsum of the left foot. He was treated with medications and referred to physiotherapy with Ms Caroline Davie.
He remained off work for two or three months. He then returned to work on light duties driving a table top truck to and from Bunnings doing deliveries, consisting of pallets of goods which were forklifted on and off the truck. He had difficulty reaching down to ratchet up the straps to secure the pallets and had to cease this work after a week. He was off work another month whilst continuing physiotherapy.
He then returned to work driving a light courier van to Murrurundi. He lasted a few months but had increasing problems with bending and lifting, which was required repeatedly with the courier parcel deliveries. His sciatica was stirred up.
He had to quit this job and then joined 13Cabs in 2021 and is still driving cabs between five and nine hours a day, up to five days per week.
He then seemed to improve until he had a recurrence of low back pain and left sided sciatica after lifting his 80-year-old father who had fallen on the floor at home on 17/8/2020. He attended Tamworth Base Hospital at the time.
The physiotherapist there would not treat him, so he attended Proactive Physiotherapy for three visits and was given a set of Therabands and shown exercises, after which he was able to self-manage his condition using the Therabands. He now uses the bands for a couple of days, up to twice a day, when his sciatica flares up and is able to control it successfully.
A few months after the accident, he developed ED (erectile dysfunction) and urgency of bladder and bowel action if he cannot make it to a toilet on time. He saw a GP in November 2020 and was referred to a urologist, Dr Smith, who gave him Cialis for the ED, but in the meantime, he lost his relationship. He also ordered a urinary ultrasound investigation but Mr Kelly was too embarrassed to proceed with this test.
In the meantime in 2019, he had been referred to a neurosurgeon, Dr Dandie, visiting Tamworth, regarding his lumbar spine condition but he did not follow through, as he didn't want to have surgery on his back.
When asked about the wrist, he says he never had treatment for the right wrist as it was manageable within a few days after the accident. He would just notice the occurrence of intermittent numbness at the volar aspect of the right wrist but not into the hand.
His right shoulder is fine. He says his knee condition affecting both knees are nothing to do with the accident. He opens his bowels when he wakes up in the morning and needs to go again half an hour after lunch. Some days he moves his bowels two or three times a day and the motions are normal.
He plays lawn bowls but uses an extendable arm to roll the bowl along the green, so he doesn't have to bend down, because of back problems. He has four schooners after bowls and the club is about 200m from his home, and he sometimes can't make it home in time to the toilet.
He takes turmeric, glucosamine and chondroitin sulphate supplement combination, one twice a day; Panadol Osteo, two tablets three times a day; and Nurofen two tablets in the morning.
He has escitalopram for his anxiety and Ramipril for high blood pressure, which he has had since age 40, allopurinol for gout and Somac for GORD.
He also takes Mirtazapine, one at night, to help him get to sleep.
He is under the care of his GP, Dr Tapim at Tamworth.
He gets recurrent exacerbations of low back pain and referred symptoms to the right lower extremity. His right wrist and right shoulder conditions have resolved. His knee conditions are not related to the accident.
He was left-hand dominant, 192cm in height and weight 138.6kg. He was of tall, solid build.
There was tenderness in the lower back centrally and to the right and left in the mid-line. There was muscle tightness bilateral paraspinal muscles.
Flexion one-half, extension one-third, lateral flexion to the right two-thirds and to the left one-half. Rotation to the right was full and to the left three-quarters of normal.
Reflexes and power in the lower limbs were normal. Plantar responses were both flexor. Sensation in the right leg was normal and in the left leg was partially reduced to light touch and pin prick in the lateral left thigh and calf to the foot in a dermatomal distribution (S1).
Supine straight leg raising was negative on the right and showed a positive stretch test on the left.
Thigh girth; right 58cm, left 57cm at 10cm above the superior patellar pole.
Left girth; right 47.5cm, left 46.5cm at 18cm below the inferior patellar pole (maximal circumference).
Right and left shoulder showed full range of movement in all six planes. There was full range of movement of both elbows, wrists and hands.
Sensation was decreased to light touch and pin prick in the ulnar border of the right hand and little finger. There was no wasting and no clawing of the hand. Grip was strong bilaterally.
Tinel's sign was negative over the ulnar nerve bilaterally at the elbow and the wrist.
Active range of movement measured with a goniometer. Right knee 0-130° of flexion with crepitus present but no patellar tenderness on compression. Left knee 0-120° of flexion with some crepitus present in the patellofemoral joint but no patellar tenderness on compression.
3/6/2019 - MRI lumbar spine - A Woodward - Desiccation and mild loss of posterior disc height at L5/S1 with small right paracentral disc protrusion abutting but not compressing or displacing the traversing right S1 nerve root. The L5 nerve roots exit normally. Note - This is not concordant with the clinical history of lower back and left-sided leg pain with numbness on the dorsum of the left foot being unable to mobilise without a stick.
27/4/2023 - MRI lumbar spine - muti-level disc height loss with reduced signal intensity in keeping with degenerative disc disease and disc desiccation. There is a mild broad based L4/L5 posterior disc bulge. With disc osteophyte complexes laterally. Appearing to contact but not displace or compress the descending left L5 nerve root. No nerve root impingement. There is a left L5/S1 posterolateral disc osteophyte complex, which appears to contact the extra-foraminal segment of the exiting L5 nerve root without evidence of displacement or compression.
(i) lumbar spine soft tissue injury, with aggravation of a pre-existing lumbar spine condition with left leg sciatica which was briefly symptomatic in 2012, but had then settled rapidly with conservative treatment;
(ii) right upper arm and forearm soft tissue injury, involving the ulnar nerve clinically, and including the right shoulder; and
(iii) possible cervical spine soft tissue injury.
The cervical spine condition resolved in short order after the accident.
Also referred were radiating symptoms down the left leg and right leg, a right shoulder soft tissue injury, intermittent bowel disturbance and bilateral knee pain.
The right ulnar nerve forearm problem is related to the accident, as this is mentioned in the Claim Form and in the early GP record.
The bilateral knee pain is not related to the accident, as there is no mention of this condition until November 2020 in the GP records, reported to have occurred after the past few weeks of truck driving after he had quit his job as a chef. Because of the 18-month delay between the mention of knee pain in the contemporaneous medical record and the accident, the accident is not considered to be a cause of bilateral knee pain or knee injury.
The radiating symptoms to the right and left leg (mainly) are the result of nerve root irritation, presumably at the L5/S1 level of the lumbar spine and are part of the lumbar spine injury and are not separate injuries.
The right shoulder soft tissue injury is considered to be caused by the accident, as it is mentioned in the early GP record.
The intermittent bowel disturbance has not been diagnosed or investigated. From talking to the claimant, it appears to be more a condition of anxiety related to lack of a readily available toilet when there is urgency of defecation, rather than being an organic disorder. There is no information available which relates the effects of the motor vehicle accident to a bowel disturbance of urgency of defaecation.
Opioid medication, which is prescribed for strong pain following accidents of this sort, would be expected to cause constipation and hypomotility of the intestinal tract rather than urgency, a symptom of hypermotility.
Lumbar spine including sciatic pain to left leg mainly and occasionally right leg is a non-threshold injury, as there are two signs of radiculopathy on clinical examination, namely dermatomal sensory changes and a positive nerve root tension sign on the left side.
As mentioned, the MRI scan report of 2019 indicates a right-sided disc lesion with the potential for right-sided nerve root irritation and the follow-up MRI scan of 2023 suggests the potential for left lower lumbar nerve root irritation..
The right forearm ulnar nerve neuritis is diagnosed as a soft tissue injury, and as referred, likely involving the ulnar nerve, making this a non-threshold injury.
The right shoulder soft tissue injury is a threshold injury. There is no indication that investigations have been performed at this part which would demonstrate a complete or partial rupture of tendons, ligaments, menisci or cartilage in the shoulder.
The lower digestive system intermittent bowel disturbance and right and left knee injuries are not causally related to the accident and therefore a determination of threshold injury is not required.
The bilateral knee condition is not related to the accident and therefore a determination of threshold injury is not required.
Lumbar spine - the presence of radiculopathy places the claimant in DRE lumbosacral impairment category III, which gives 10% WPI.
Although there was a prior history of sciatic symptoms in the left lower extremity, representing non-verifiable radicular complaints, and therefore a potential DRE category II giving 5% whole person impairment seven years before the index accident, there was no evidence that this condition was symptomatic at the time of the index accident. Hence there are no grounds for subtracting a deduction from the assessed permanent impairment.
Right ulnar nerve neuritis - There is decreased sensibility, with or without abnormal sensation or pain forgotten during activity, giving the claimant a grade 2 sensory deficit of the ulnar nerve above mid forearm. This is assessed as 25% of the maximum sensory deficit. This ulnar nerve sensory condition gives a maximum of 7% upper extremity impairment, at either above or below mid-forearm levels. 25% of 7% is 1.75%, rounded to 2% upper extremity impairment. 2% upper extremity impairment is equivalent to 1% whole person impairment.
Right shoulder-There was full range of movement in all six directions at the right shoulder, hence no assessable permanent impairment.
The combined whole person impairment, arising from injuries related to the accident: 10% combined with 1% giving 11% whole person impairment.
References: AMA4 chapter 3, table 72, p110; table 3, p20, table 11, p48, table 15, p54: combined values chart. Motor Accident Guidelines 6.138, 6.31.
[4] Briggs v IAG Limited t/as NRMA Insurance [2022] NSWSC 372.
[5] Insurance Australia Limited t/as NRMA Insurance v Trkulja [2023] NSWSC 956.
[7] Allianz Insurance Australia Group Limited v Keen [2021] NSWCA 287 and Allianz Insurance Australia Group v Marsh [2021] NSWCA 31.