austlii:nsw/NSWPICMP/2026/200
General information, not legal advice.
NSWPICMP
2026-03-17
Original judgment source is linked above.
Insurance Australia Limited t/as NRMA Insurance v Kang [2026] NSWPICMP 200
Insurance Australia Limited trading as NRMA Insurance
MOTOR ACCIDENTS - Motor Accident Injuries Act, 2017; Medical Review Panel; lumbar spine; cervical spine; right shoulder; whole person impairment (WPI); inconsistency; causation; claimant suffered injury in a rear end collision on 1 June 2022; Medical Assessor (MA) certified 17% WPI for injury to lumbar spine; injury to cervical spine; injury to right shoulder; insurer sought review; Held - inconsistencies in presentation; Briggs v IAG Limited Trading as NRMA Insurance applied; claimant sustained injury to cervical spine, lumbar spine and SLAP lesion of the right shoulder caused by the accident; lumbar spine assessed as DRE category I or 0% WPI; cervical spine assessed as DRE category II or 5% WPI; due to inconsistency right shoulder assessed by analogy at 2% WPI; certificate of the original MA revoked, total assessed at 7% WPI.
Whether the degree of permanent impairment of the injured person as a result of the injury caused by the motor accident is greater than 10%
Certificate issued under s 7.23(1) of the Motor Accident Injuries Act 2017
14 August 2025 and determines that the accident was a cause of the following injuries which give rise to a permanent impairment of 7%: * cervical spine - soft tissue injury; * lumbar spine - soft tissue injury, and * injury to the right shoulder - slap tear.
non-economic loss unless the degree of permanent impairment of the injured person as a result of the injury caused by the accident is greater than 10%. 5. This dispute is in relation to whether the degree of permanent impairment sustained by
Mr Kang as a result of the injury caused by the accident is greater than 10%. This constitutes a medical assessment matter pursuant to Schedule 2, cl 2 of the MAI Act. 6. A medical assessment matter is determined in accordance with Division 7.5 of the MAI Act by a Medical Assessor.[1] 7. The dispute as to permanent impairment in respect of the claimant's physical injuries was referred to Medical Assessor Shahzad. He issued a certificate dated 14 August 2025.
18 December 2025 the claimant uploaded an Application to Lodge Additional Documents with attachments (ALAD).
"6.6 Causation is defined in the Glossary at page 316 of the AMA4 Guides as follows:
'Causation means that a physical, chemical or biologic factor contributed to the occurrence of a medical condition. To decide that a factor alleged to have caused or contributed to the occurrence or worsening of a medical condition has, in fact, done so, it is necessary to verify both of the following:
This, therefore, involves a medical decision and a non-medical informed judgement.
6.7 There is no simple common test of causation that is applicable to all cases, but the accepted approach involves determining whether the injury (and the associated impairment) was caused or materially contributed to by the motor accident. The motor accident does not have to be a sole cause as long as it is a contributing cause, which is more than negligible. Considering the question 'Would this injury (or impairment) have occurred if not for the accident?' may be useful in some cases, although this is not a definitive test and may be inapplicable in circumstances where there are multiple contributing causes."
Certificate of Medical Assessor Wijetunga dated 24 August 2023
"The Panel notes there has been a consistent history of pain and restriction of movement following the accident and the MRI scan shows a SLAP lesion which is a superior labral tear. Whilst the MRI scan shows that the rotator cuff mechanism is intact the Panel finds the SLAP tear is in keeping with the claimant's symptoms and restriction of movement."
21 August 2020, and * on 21 February 2022 the claimant was stressed, collapsed at work and was taken to hospital for vestibular neuritis. 8. The insurer relies upon the decision of the First Review Panel to assert that the annular tear and the changes shown on the MRI scan of the lumbar spine were pre-existing and not caused by the accident. 9. The insurer relies upon a Collision and Biomechanical Report of Dr Andrew McIntosh dated 22 October 2024. Dr McIntosh reported that pre-existing injury is a risk factor in injury likelihood and symptom presentation following a low severity car crash. He opined the closing speed was likely less than 15 kmph and that the claimant's injuries were not consistent with what he considered to be the likely biomechanical forces in the accident. He concluded there was no mechanism for shoulder injury where there was no direct or indirect blunt force loading of the shoulders in the incident. He concluded there was a very low likelihood of soft tissue injury of the cervical spine within the crash severity of the collision. Further the thoraco-lumbar spine would have been well supported by the seat and seat belt and the loads applied to the spine would have been very low magnitude and unlikely to cause injury. 10. The insurer relies upon the opinion of Dr Machart who assessed 0% WPI as a result of the accident. He found a strong element of pain behaviour and even accepting there were injuries to the cervical and lumbar spine he concluded they would be soft tissue injuries and attract a 0% WPI.
Dr Lee of 3 June 2022 show that the shoulder is shaded as one of the body parts in which the claimant felt pain. Dr Lee also recorded restriction of motion. 3. Furthermore, consistent with the opinion of Dr Chieng the claimant submits that when he saw Dr Lee for a follow up review on 11 June 2022 his shoulder was listed as a body part in which he feels pain, and it was rated as 9 on a scale of 10. 4. The claimant submits that he began experiencing pain and a restricted range of motion in his right shoulder from right after the accident. The claimant submits the opinion of
Dr Herald should be preferred to the opinion of Dr Machart. 5. In relation to the cervical and lumbar spine the claimant submits the opinion of Dr Herald should be preferred to the opinion of Dr Machart. 6. In relation to the prior accident it is submitted the claimant had fully recovered and therefore should not be subject to a deduction for a pre-existing symptomatic permanent impairment. 7. When considering the report of Dr McIntosh, the claimant submits as per Insurance Australia Limited T/as NRMA Insurance v Mangogna the contents of that report must be weighed against the rest of the existing evidence.[13] 8. The claimant provided submissions dated 7 October 2024 in response to the insurer's application for review.[14] Those submissions address the question of whether Medical Assessor Shahzad was incorrect in a material respect in his certificate and do not address the substantive issue to be determined by the Panel.
"On the red light, [1], [2], [3] cars were stopped. Mine was [2] and there was [1] in front of mine, [3] was behind of mine. All the sudden, I felt my back part of car was hit and felt pain on my neck and back. Someone was knocking my door glass and that was driver of [4]. [4] didn't stop the car on red light, so [4] hit [3], then [3] hit mine and I hit [1]. The driver of [1] just left as he thought his car looked okay. I exchanged detail with [3] & [4]. Next day I felt worse on my neck and back. I contacted to my insurer to fix my car and met my GP."[15]
"I feel hard to move my neck. I feel pain on my back. I feel numbness in my hand."
whilst driving - had tingling L sciatic distribution
"Neck/trapezius/Whole Back/R+L hip/pelvis/ anterior neck and upper chest/. Injuries to be further reviewed.
16 November 2022 the additional notation appears "SLAP lesion MRI / R shoulder".[25] 6. On 11 June 2022 Dr Lee circled areas to the back and sides of the neck, the upper and lower thoracic spine and lumbar spine on a diagram.[26] 7. On 9 October 2022 Dr Kwak reported ongoing paraesthesia in the left buttock to the toe and lightly reduced left ankle reflex. He was referred to the Royal Prince Alfred Hospital Neurosurgical Clinic for review. 8. Dr Lee provided a report dated 6 May 2023. In relation to the cause of injury to the right shoulder he stated:
"- During the collision his right shoulder muscles must be suddenly and forcefully contracted against sudden relative movements between his body and the vehicle.
- This can torn labrum by pulled labrum (SLAP tear) forcefully [sic]
- He did not had any shoulder injuries prior to the accident
- His right shoulder injury was a direct cause of the motor vehicle accident
- On initial presentation on 2/6/22 he reported 'shoulder pains on a diagram provided and physical examination performed on the same day reported shoulder pains'( ...).
- In initial certificate these seems to be interpreted as trapezius pain and cervical radicular pathologies. Shoulder pains, trapezius pains and cervical radicular pains are often mimics each other."[27]
"He was hit from behind whilst standing at a traffic light. He developed pain symptoms in his neck, shoulder girdle and back. He has been experiencing left forearm paraesthesia and some vague radicular symptoms in his left lower limb. He has ongoing left side lower back pain, neck pain and shoulder girdle pain symptoms."[28]
"... the sudden acceleration and deceleration of the vehicle and the forceful contraction of his shoulder muscles would have caused injury. The supraspinatus contraction causing tendinitis while forceful biceps contraction would have pulled the labrum causing the tear."
"Mr Kang has torn his right shoulder labrum. This is a specialized cartilage histologically similar to a meniscus. There are both made up for fibrocartilage. As per the definition of a 'minor injury', a torn meniscus or labrum does not fit in with a 'minor injury'.
Furthermore, Mr Kang also has rotator cuff tendinosis. Tendinosis. Tendinosis is basically small multiple tears of a tendon that cannot be clearly visualized as opposed to a full thickness tear. It is usually the precursor to a visible tear if it does not heal. Again, tendon tears do not count as 'minor injuries'".
"At L4/5. A diffuse disc bulge combined with mild facet arthrosis leads to mild central canal narrowing and potential irritation of the descending L5 nerve roots. No further central canal narrowing is evidence and there is no definite evidence of neural impingement."[34]
Findings: Lumbar spine normal in alignment. No fracture or bony lesion is demonstrated. Conus terminates at L1. Conus and cauda equina are normal in appearance.
L2/3: Disc signal and height is maintained. There is subtle anterior annular fissuring with mild bone stress response. No posterior disc pathology or mechanical neural impingement.
L3/4: Disc signal and height is maintained. There is anterior annular fissuring with mild bone stress response. No posterior disc pathology is demonstrated. No foraminal or canal stenosis.
L4/5: Disc dehydration and mild concentric disc bulging. No focal disc protrusion is demonstrated. Mild anterior annular fissuring with mild bony stress response. Annular fissuring at L4/5 with disc bulge contacts the left L5 nerve root in the lateral recess. Mild bilateral foraminal stenosis and mild canal stenosis. Early right facet joint arthritis without bone stress or synovitis.
L5/S1: Mild loss of disc height. Mild posterior disc bulging. Central posterior annular tear without focal disc protrusion. No mechanical neural impingement is demonstrated. Early bilateral facet joint arthritis without synovitis or bone stress.
Conclusion: Mild annular fissuring demonstrated at the lower four lumbar levels. No focal disc protrusion. Early disc degeneration L4/5 and L5/S1.
Annular fissuring at L4/5 with disc bulge contracts the left L5 nerve root and would be a potential site of irritation as a cause for left sciatica...."[35]
MRI lumbar spine, 10 August 2022 incorrectly dated as 1 June 2022
"Technique: MRI assessment has been performed utilising sagittal T1, T2 STIR sagittal T2, T2 FS sequences on the 1.5 Tesla Siemens MRI unit.
Findings: Lumbar spine alignment shows mild reduced lordosis. Lower thoracic cord, conus medullaris and cauda equina nerve roots are normally distributed. No fracture or marrow contusion is seen.
Assessment of the individual disc levels shows at L1/2, L2/3 and L3/4 no disc protrusion or neural compression.
L4/5 shows disc dehydration. A small disc bulge is noted without neurocentral compression. There is mild foraminal stenosis. Left-sided extraforaminal annular tear is evident. Facet joints do show mild facet joint effusions.
L5/S1 shows disc desiccation and a small subligamentous disc bulge with posterocentral annular tear. No central canal compromise is seen. Exiting nerves pass freely. Facet joints are mildly arthritic.
No pars defects are seen and paraspinal soft tissues are normal.
Conclusion: MRI of the lumbar spine does demonstrate at L4/5, a broad-based disc bulge with a left extraforaminal annular tear. Mild foraminal stenosis is seen abutting but not deviating the existing L4 nerve roots. L5/S1 is desiccated with a posterocentral annual tear. Mild facet joint arthropathy is seen."[36]
"Findings: Subacromial/subdeltoid bursa shows a small volume of fluid. Assessment of the rotator cuff shows and intact teres minor tendon. Infraspinatus shows mild articular fraying and low-grade enthesopathic cyst at the attachment. Supraspinatus is intact. No cuff tear is seen. There is no denervation or atrophy.
There is no deltoid dehiscence. Coracoacromial ligament is intact. AC joint is intact.
Long head of biceps is normally located with intact bicipital anchor. Subscapularis is normal.
Assessment of the labrum and capsule demonstrates signal hyperintensity of the anterosuperior labrum and the labrum superiorly across the 12-o'clock position extending to the posterior labrum suspicious for a SLAP tear. Articular surfaces are maintained. Capsule is intact. No fracture is noted. Axillary contents are normal.
Conclusion: MRI of the right shoulder does demonstrate signal hyperintensity of the labrum anterosuperiorly across the bicipital anchor in keeping with a SLAP tear without destabilisation of the bicipital anchor. No cuff tear is noted."[37]
"No bony injury or bone stress of the left hemipelvis or left hip.
Minimal spurring along the lateral margins of the acetabular roof from 12 to 10 o'clock. No degenerative change of the left hip, otherwise preserved cartilage throughout.
Subtle oedema of the trochanteric bursal space, borderline for trochanteric bursitis."[38]
"Moderate cartilage wear of the right glenohumeral joint. There is thickening of the glenohumeral joint capsule at the axillary recess. There is also oedema within the rotator interval. These findings can be seen adhesive capsulitis.
There is a fairly large superior labral tear from approximately 3 o'clock to 9 o'clock and there does appear to be fraying and possible small tear at the biceps anchor at the insertion, but without flipped fragment or bucket handle tear.
Rotator cuff tendinopathy with low-grade partial supraspinatus tendon tear anteriorly."[39]
28 August 2024.[40] 2. Dr Machart reported the claimant informed me he had no physical issues before the accident. He reported neck pain, low lumbar pain and right shoulder pain and stiffness. 3. In respect of the lumbar spine, he reported the claimant was tender on the lower half of the lumbar spine, there was no spasm, movement was diminished by a third and radicular signs were not evident. 4. In respect of the cervical spine he reported tender paraspinal muscles, movement reduced by a third and reportedly painful at extremes. There was no asymmetry, no radicular symptoms, no spasm and no deformity. 5. In respect of the right shoulder not reported tenderness and withdrawing. All movements were reported to be painful and diminished including the asymptomatic left shoulder. He reported the following range of motion:
Report of Dr Andrew McIntosh biomechanical engineer
"I have considered his injury mechanism, which also includes sudden/forceful contraction of shoulder muscles. I agree that a driver might contract the shoulder muscles. No acute injury was included in the initial certificates of capacity, including a dislocation or subluxation. In my opinion, the actual forces acting through the Claimant's right shoulder, including any forces related to muscle contraction, would have been low magnitude and unlikely to cause a SLAP tear or other right shoulder musculoskeletal disorder."
6 December 2019. This details some back pain with tingling in a left sciatic distribution.
Mr Kang stated he could not recall this. He specifically denies any prior problems with his neck, back or shoulders in the period immediately before the accident in June 2022.
History of symptoms and treatment following the accident
27 February 2026 to consider its determination. 2. In considering causation the Panel refers to the test for causation set out in the Guidelines. However, the Panel also notes in Briggs v IAG Limited Trading as NRMA Insurance Wright J reminded us that the relevant legal test in relation to causation does not require scientific certainty.[43] His Honour stated at [70]-[72]:
"70. This reasoning does not accord with the relevant legal test in relation to causation, which does not require scientific certainty. In Metro North Hospital and Health Service v Pierce [2018] NSWCA 11, the Court of Appeal said, in relation to causation in a similar context, as follows at [138] (White JA, Macfarlan and Payne JJA agreeing):
'138 Whether the Hospital's negligence in not responding to the induced seizures in a timely manner materially contributed to Ms Pierce's worsened condition is not to be determined on the basis of scientific certainty, but on the balance of probabilities. As Spigelman CJ said in Seltsam Pty Ltd v McGuiness (2000) 49 NSWLR 262; [2000] NSWCA 29 at [143]:
'An inference of causation for purposes of the tort of negligence may well be drawn when a scientist, including an epidemiologist, would not draw such an inference'.'
'... it is not incumbent upon the applicant, upon whom the onus rests, to produce evidence from medical witnesses which proves to demonstration that the applicant's contention is correct. Medical science may say in individual cases that there is no possible connexion between the events and the death, in which case, of course, if the facts stand outside an area in which common experience can be the touchstone, then the judge cannot act as if there were a connexion. But if medical science is prepared to say that it is a possible view, then, in my opinion, the judge after examining the lay evidence may decide that it is probable. It is only when medical science denies that there is any such connexion that the judge is not entitled in such a case to act on his own intuitive reasoning. It may be, and probably is, the case that medical science will find a possibility not good enough on which to base a scientific deduction, but courts are always concerned to reach a decision on probability and it is no answer, it seems to me that no medical witness states with certainty the very issue which the judge himself has to try.'
Dr McIntosh, biomechanical engineer who noted a proportion of vehicle occupants may suffer a whiplash associated disorder/soft tissue injury from a rear end collision. Therefore, the first component of causation is met in respect of injury to the cervical spine. 2. Whilst Paul Monaro, physiotherapist reported on 11 June 2020 that the claimant had attended eight sessions of treatment for his neck, lower back and left leg he also reported that Mr Kang continued to complain of numbness associated with the right foot and low back discomfort. 3. One day after the accident Dr Lee, GP reported complaints of severe neck pain and in the Application for personal injury benefits completed on 3 June 2022, Mr Kang reported he felt worse on his neck and back the day after the accident. On 3 June 2022 Mr Kang commenced treatment with Mr Oh, physiotherapist to the left side of the neck, the upper back and in respect of pain radiating down the left upper limbs and the left lower limbs. Thereafter there were consistent complaints of pain and restriction of movement of the cervical spine and Medical Assessor Barnsley reported Mr Kang asserted his neck pain had persisted since the accident. 4. Given Dr McIntosh, biomechanical engineer noted a proportion of vehicle occupants may suffer a whiplash associated disorder/soft tissue injury from a rear end collision and having regard to the contemporaneous and consistent complaint of symptoms relating to the cervical spine the Panel is satisfied that the accident did cause or contribute to the injury sustained by the claimant to the cervical spine. 5. On examination Mr Kang had complaints of neck pain without evidence of dysmetria, guarding or spasm. There were neurological complaints in the upper limbs but no clear evidence of radiculopathy. The Panel finds the claimant sustained a soft tissue injury to the cervical spine caused by the accident.
Dr Kwak reported a history of back pain on and off, and on examination concluded
Mr Kang had muscle spasm with underlying sciatica. A CT scan of the lumbosacral spine the same day reported a diffuse disc bulge at L4/5 combined with mild facet arthrosis leading to mild central canal narrowing and potential irritation of the descending L5 nerve roots. 5. Following a motor vehicle accident in March 2020 Mr Kang reported he twigged his back and on 3 April 2020 Dr Kwak recorded ongoing lower back pain and spasm radiating to the buttocks and hamstrings. 6. On 12 June 2020 and 24 July 2020 Dr Kwak noted restriction of movement, paraesthesia, reduced reflexes and reduced sensation at the L4/5 level. Mr Kang had a left L5 perineural steroid injection on 21 August 2020. On 9 October 2020 Dr Kwak reported paraesthesia from the left buttock to the toe and a reduced left ankle reflex. 7. The Panel notes there is no reported complaint relating to the lumbar spine between October 2020 and the accident on 1 June 2022. 8. The accident occurred on 1 June 2022 and on 2 June 2022 Dr Lee noted injury to the whole back. In his certificate of capacity/certificate of fitness dated 2 June 2022 Dr Lee identified the claimant's history of previous lumbar disc pathology but noted the accident caused new and extended symptoms. 9. In the Application for personal injury benefits dated 3 June 2022 the claimant described pain in his back. 10. On 10 July 2020 the claimant underwent an MRI of the lumbar spine which reported disc dehydration and mild concentric disc bulging, but no focal disc protrusion. However, annular fissuring at L4/5 with the disc bulge was said to contact the left L5 nerve root. Similar findings were made in an MRI of the lumbar spine of 10 August 2022 which demonstrated a broad-based disc bulge at L4/5 with a left extraforaminal annular tear. 11. The claimant was reviewed by Dr Bak, rheumatologist on 22 October 2022. He reported there were no radicular features present and noted the MRI of the lumbosacral spine hadn't changed much since 2020. 12. The Panel notes the history of prior injury associated with clinical findings of back pain and left sided sciatica. Given the MRI scan of 10 July 2020 prior to the accident showed damage at the L4/5 level which was unchanged at the time of the post-accident MRI scan of 10 August 2022 the Panel is of the opinion, the annular tear and the changes shown on the MRI scan were pre-existing and not caused by the accident. The Panel concurs with the view of the First Review Panel in concluding that the signs of radiculopathy were present pre-accident and are not causally related to injury sustained in the accident. 13. Notwithstanding the opinion of Dr McIntosh and given the legal test for causation does not require scientific certainty the Panel, having regard to the consistent history of complaint post-accident and the opinion of Dr Lee that the accident caused new and extended symptoms, finds the accident caused the claimant to sustain a soft tissue injury to the lumbar spine.
(a) pain with guarding; or
(b) non-uniform range of motion - dysmetria; or
(c) non-verifiable radicular complaints defined in table 6.8 as
(i) symptoms (shooting pain, burning sensation, tingling), and
(ii) which follow the distribution of a specific nerve root but no objective clinical findings such as loss or diminished sensation, loss of diminished power or loss or diminished reflexes.
Table 19 on page 59 and Table 18 on page 58 AMA 4 Guides
[13] Insurance Australia Limited T/as NRMA Insurance v Mangogna [2023] NSWPICMP 588.
[43] Briggs v IAG Limited Trading as NRMA Insurance [2022] NSWSC 372.
[44] Bath v Allianz Australia Insurance Limited [2026] NSWSC 165 at [21] - [22].
[45] Allianz Australia Insurance Limited v Bell [2025] NSWCA 187 at [105]- [111].
Insurance Australia Limited t/as NRMA Insurance
Kang [2026] NSWPICMP 200;
(2000) 49 NSWLR 262