austlii:nsw/NSWPIC/2025/639
General information, not legal advice.
NSWPIC
2025-12-01
Original judgment source is linked above.
30 October 2025, Mr Edward McMahon, counsel, appeared for the applicant, instructed by Turner Freeman Lawyers. Mr Boris Necovski, counsel, appeared for the respondent, instructed by Turks Legal. 4. I am satisfied that the parties to the dispute understand the nature of the application and the legal implications of any assertion made in the information supplied. I have used my best endeavours in attempting to bring the parties to the dispute to a settlement acceptable to all of them. I am satisfied that the parties have had sufficient opportunity to explore settlement and that they have been unable to reach an agreed resolution of the dispute.
(a) whether the treatment is reasonably necessary treatment as a result of the injuries, and the extent and quantification of the applicant's entitlement to payment of medical and related expenses pursuant to s 60 of the 1987 Act.
(a) ARD and attached documents;
(b) Reply to ARD and attached documents, and
(c) Application to Lodge Additional Documents filed on behalf of the applicant on
27 October 2025.
(a) following an injury in 2005, he then underwent an L5/S1 laminectomy and disc excision and rhizolysis of the S1 nerve root, which led to an improvement in symptoms and him being able to continue working full-time in physical jobs without any until the accepted injury in March 2022;
(b) on 1 March 2022, he sustained the accepted injury when his backside and lower back were jolted as the wheels of a forklift that he was driving fell into an excavated hole which had been left uncovered;
(c) he experienced severe backpain at the time of the accepted injury and subsequently developed worsening back, left leg weakness and left sided foot drop;
(d) he sought medical treatment and underwent multiple procedures for those symptoms which included: total left hip replacement on 25 September 2023; L4/5 and L5/S1 anterior interbody fusions and pedicle screw fixation on
18 January 2024; an L4/5 and L5/S1 anterior lumbar interbody fusion on
27 May 2024; a left L5 nerve root decompression on 25 June 2024;
(e) he continues to experience persistent and worsened left foot drop and worsened numbness in his left foot and toes to the extent that he now has almost no feeling on his left foot and toes;
(f) he is also distressed by ongoing lower back pain, which radiates down into his left leg in a lightning-bolt type manner and he also now also experiences intermittent right leg pain, tingling and numbness;
(g) since developing his left foot drop, he regularly stumbles and trips and he is now heavily reliant on the use of a walker and/or walking stick and is now severely restricted in his ability to go out and to shower and dress, and
(h) he wishes to undergo the treatment as he believes it will provide him with relief and improve his quality of life.
(a) in the early days after his accepted injury, he tried a splint to assist with his left foot weakness, however he found it to be uncomfortable and unsupportive and his foot still twisted and felt very unstable;
(b) his work boots provided more support than the splint, but he still experiences instability, and
(c) he is now very reliant on a walker and/or walking stick for mobility.
(a) between November 2001 and October 2006, Professor Youseff Ghabrial, orthopaedic and spinal surgeon, issued various reports in relation to ongoing back pain with right leg symptoms, and involving his cervical spine and lumbar spine, which the applicant developed since 1992 and that the applicant had an aggravation at work on 21 December 2004. Professor Ghabrial noted that the applicant had right L5/S1 disc prolapse with sequestration. On 21 June 2025, Professor Ghabrial performed L5/S1 laminectomy, and rhizolysis of the S1 nerve roots, which diminished the applicant's problems although the applicant had some residual symptoms in his back and legs, including cramping and pins and needles in his left leg. Professor Ghabrial attributed the applicant's condition and surgery to aggravation at work on 21 December 2024 and assessed 14% total whole person impairment as a result of the injuries sustained on
21 December 2004;
(b) on 7 December 2021, an X-ray of the left foot, was reported to show degenerative changes of multiple joints;
(c) on 8 December 2021, the applicant was treated at Wyong Hospital in relation to left ankle pain and pain in his lower limb and hip;
(d) on 11 April 2022, an X-ray of the left foot, was reported to show mild degeneration of multiple joints;
(e) on 14 April 2023, an X-ray of the hips, knees, left ankle, was reported to show advanced bone-on-bone arthropathy of the left hip with bony remodelling and likely impingement;
(f) on 14 April 2023, a CT scan of the lumbosacral spine was reported to show spinal canal compromise in the lower lumbar spine, with facet arthropathy most severe at L4/5 and foraminal compromise most severe at L5/S1;
(g) on 15 July 2023, Dr Coughlan, treating neurosurgeon, reported that, after having worked in construction for many years involving heavy lifting, the applicant had developed progressively worsening leg pain and back pain, a severe antalgic gait, he could not support himself on his left leg and had a reduction in hip flexion on the left side. Dr Coughlan noted that the applicant had significant degenerative changes in the lumbar spine but noted that the main issue seemed to be his left hip with severely denuded left lift hip joint with bone on bone. Dr Coughlan stated that he thought the applicant may require a decompression in his lumbar spine although the priority was his severe left hip pain which required a hip replacement. Dr Coughlan noted that the applicant would get an MRI of the lumbar spine;
(h) on 20 July 2023, Dr Michael Hunter, orthopaedic surgeon, reported a history that following the accepted work injury, the applicant had gradually progressive left leg pain, functional loss and weakness as a result of severe left hip osteoarthritis and L5/S1 compression in his spine. Dr Hunter reported that on examination the applicant's hip demonstrated no range of movement in flexion, severe irritability and was fixed in a rotated position which made gait difficult and that the applicant walked with a severely antalgic abductor gait. Dr Hunter noted that a radiography of the applicant's left hip showed severe chondral loss, bony loss, deformity present and spur function and that a CT scan of his back showed significant L5/S1 lateral recess problems, which corresponded with the applicant's neurological loss in his lower limbs. Dr Hunter recommended that the applicant undergo hip replacement surgery;
(i) on 21 July 2023, an MRI of the lumbar spine was reported to show mild to moderate central canal stenosis at L4/L5 and degeneration of the bilateral L4/L5 and L5/S1 facet joints, right central disc osteophyte complex at L5/S1 and mild contact with the right S1 nerve root;
(j) on 9 August 2023, an X-ray of the left foot and ankle was reported to show mild osteoarthritic changes and clawing of toes;
(k) on 25 August 2023, Dr Coughlan reported that the applicant had very significant left leg pain and had mild to moderate central canal stenosis at L4/5 and some stenosis at L3/4 with some mild lateral recess stenosis. Dr Coughlan recommended a left L3/4 and L4/5 lateral recess decompression;
(l) on 25 September 2023, an X-ray of the left hip was reported to show a total joint replacement in position with no complication seen;
(m) on 11 November 2023, Dr Coughlan reported that the applicant was doing well after undergoing hip replacement, although he still had left-sided foot drop.
Dr Coughlan recommended that, as a priority, the applicant undergo decompression. Dr Coughlan noted that the applicant already had approval for L4/5 and L3/4, and he also sought approval for decompression of L5/S1.
Dr Coughlan stated that the decompression was to improve the nerve function especially on the left-hand side, and that it was not aimed to improve his axial back pain. Dr Coughlan noted that the applicant had some narrowing at L5/S1 with effacement of the S1 nerve root. Dr Coughlan stated that the applicant may later also require a procedure to put spacers at L5/S1 and L4/5 to address the collapse of the disc and the foraminal stenosis;
(n) on 18 January 2024, Dr Coughlan reported that the applicant underwent L3/4, L4/5, L5/S1 decompression;
(o) on 1 March 2024, a CT scan of the lumbosacral spine was reported to show similar interval appearances to previous scans except for findings of interval left-sided laminectomy and improvement of the spinal stenosis;
(p) on 1 March 2024, Dr Coughlan reported that the applicant had noticed significant reduction of leg pain since his decompression. Dr Coughlan noted that the applicant did still have quite marked foot drop on the left side and he had noted that the leg seemed to be rolling over and had weakness in dorsiflexion.
Dr Coughlan noted that the CT scan demonstrated chronic bilateral foraminal stenosis affecting L5/S1 however the applicant only really had motor deficit on the left side. Dr Coughlan sought an updated CT scan;
(q) on 22 March 2024, Dr Coughlan noted that the applicant was having ongoing issues with long-standing foot drop. Dr Coughlan noted that the applicant previously underwent a decompression, and expressed concern that some of the compression of the nerve was in the foramen, although curiously the applicant was not rolling his foot and falling on the right despite having bilateral foraminal narrowing. Dr Coughlan recommended that the applicant undergo nerve conduction studies to determine whether it was primarily a radicular issue or whether he also had issues related to his common peroneal nerve down stream which could contribute towards his left leg weakness;
(r) on 17 April 2024, Professor Robert Heard, neurologist, reported on nerve conduction studies, and concluded that there is no evidence of focal peroneal nerve lesion at the fibular head although an incomplete left sciatic trunk injury was not excluded. Professor Heard recommended a neurology review;
(s) on 26 April 2024, Dr Coughlan reported that nerve conduction studies showed that the applicant had bilateral L5 issues and no significant common peroneal nerve compression. Dr Coughlan noted that the applicant's gait had deteriorated, he was becoming more incapacitated, his foot drop was quite severe on the left and was becoming a falls risk, and the applicant was also getting significant issues on the right side. Dr Coughlan recommended an L4/5 and L5/S1 anterior interbody fusion with posterior pedicle screws and wide foraminotomies at both levels with partial facetectomies to address the foraminal compromise;
(t) on 27 May 2024, Dr Coughlan reported that the applicant underwent an L4/5 and L5/S1 anterior lumbar interbody fusion;
(u) on 28 May 2024m Dr Coughlan reported that the applicant underwent L4/5 and L5/S1 posterior pedicle screw fixation;
(v) on 30 May 2024, a CT scan of the lumbar spine was reported to show moderate to severe left foraminal stenosis at L5/S1 with loss of the normal fat plane surrounding the nerve root suggesting left L5 nerve impingement. Moderate left foraminal narrowing at L4/5 potentially impinging the left L4, moderate to severe canal stenos at L3/L4 level, a right posterior dominant disc osteophyte complex, with narrowing of the right subarticular recess at L5/S1 and severe right foraminal stenosis;
(w) on 7 June 2024, an MRI of the lumbar spine was reported to show no complicating features post fusion of L4 to S1, mild diastasis of bilateral L4/5 facet joints with effusion, right posterior osteophyte at L5/S1 causing lateral recess stenosis and potential impingement of right S1 nerve and mild constitutional lumbar canal stenosis;
(x) on 13 June 2024, Dr Coughlan reported that he was concerned with the applicant's ongoing foot drop which had not improved. Dr Coughlan noted that the MRI scan showed the L4/5 facet rather diastased, which Dr Coughlan suspected may be causing some ongoing compression of the left L5 nerve root within the L4/5 lateral recess. Dr Coughlan stated that, given the applicant's ongoing foot drop, it was possible that that was a consequence of the longevity of his nerve compression but, for the sake of thoroughness, he suggested urgently re-exploring the L5 nerve on the left side, focusing on the L4/5 interspace and lateral recess and also the L5/S1 foramen;
(y) on 25 June 2024, Dr Coughlan reported that the applicant underwent re-exploration of the L5 nerve on the left side, focusing on the L4/5 interspace and lateral recess and also the L5/S1 foramen;
(z) on 18 July 2024, a CT scan of the lumbar spine was reported to show no evidence of hardware failure following the posterior and anterior fusion, with progressive left L3 extra foraminal compression;
(aa) on 9 August 2024, Dr Coughlan reported that, since the two-level anterior fusion with posterior screws, the applicant had noticed a marked improvement in his back pain, however he continued to suffer from persistent left-sided foot drop and very weak tibialis anterior, which had been a long-standing issue. Dr Coughlan requested an updated MRI. Dr Coughlan stated that the applicant may have some lateral recess stenosis at the level above his fusion at L3/4;
(bb) on 12 August 2024, an MRI of the lumbar spine was reported to show: at the L3/L4 level, diffuse posterior disc bulge with mild thecal sac compression and bilateral mild foraminal encroachment due to bulging disc and facet arthropathy; at the L4/L5 level, prior fusion and left hemilaminectomy, possibly a small synovial cyst arising from the left facet joint that may encroach on the left exit foramen causing mild to moderate foraminal encroachment without neural compromise, with no significant right foraminal encroachment seen; at the L5/S1 level, posterior disc osteophytic changes with minimal canal encroachment, mild to moderate foraminal encroachment and mild lateral recess stenosis with potential minimal encroachment of the descending S1 nerve roots (right more than left), which were similar to the previous study;
(cc) on 16 August 2024, Dr Coughlan reported that recent MRI showed that there may be some evidence of L3/4 stenosis and that compression could account for the applicant's persistent foot drop. Dr Coughlan recommended that the applicant undergo nerve conduction studies to exclude a common peroneal nerve palsy before they consider surgical options;
(dd) on 27 September 2024, Professor Heard, neurologist, reported that findings of a further nerve conduction study were very similar to the previous study of April 2024, with the left tibial motor response being absent, with preserved sural sensory responses. Professor Heard concluded that the findings were more in keeping with lumbar nerve root disease, although incomplete sciatic nerve lesion could not be entirely excluded;
(ee) on 4 November 2024, Dr Coughlan reported that the applicant had persistent left-sided foot drop and no improvement in strength, although his pain had improved. Dr Coughlan noted that an MRI scan showed a synovial cyst at L4/5 potentially irritating the left L4 nerve root. Dr Coughlan stated that it was difficult to know whether the lack of improvement was from a chronic neuropraxia and will not improve or whether there was still an ongoing issue affecting the left L4/L5 nerve. He noted that the L5 nerve looked very free and capacious, however on the left L4 nerve there was a far lateral cyst compressing left L4 nerve root. Dr Coughlan recommended and requested a stereotactic guided left L4/5 stereotactic guided bilateral foraminotomy to decompress the nerve in the far lateral;
(ff) also on 4 November 2024, Dr Coughlan estimated that his costs to perform stereotactic guided left L4/5 far lateral foraminotomy was $8,160;
(gg) on 3 February 2025, Dr Coughlan reported that, whilst the applicant had minimal back pain, he was concerned regarding the applicant's chronic foot drop and the atrophy in the gastrocnemius and tibialis anterior, which was impacting the applicant's overall gait and instability. Dr Coughlan recommended getting nerve conduction studies as well as an EMG and review by Professor Heard;
(hh) on 5 March 2025, Professor Heard, neurologist, who noted a history that the applicant reported that his left foot drop appeared subacutely after the accepted injury and slowly worsened, and that three nerve conduction studies had shown stable findings, with no focal abnormality of the left common peroneal nerve and the sural response was preserved. Professor Heard noted that nerve conduction studies were again unchanged from the previous studies. On examination, Professor Heard noted marked wasting of the left tibialis anterior and mild wasting of the posterior left calf muscles and severe weakness of left ankle dorsiflextion and inversion and mild weakness of left ankle plantar flexion, reduced left knee jerk, absent left ankle jerk, and loss of feeling over the left hallux and hypersensitivity over the lateral calf indicative of L5, although the right leg and upper limbs examined normally. Professor Heard opined that the findings are consistent with radicular pathology, mainly at L5 but also including L4 and S1 on the left, with a differential diagnosis of a high sciatic trunk lesion involving selectively the peroneal component of the sciatic trunk;
(ii) on 6 March 2025, Professor Heard, neurologist, reported that recent further nerve conduction studies were very similar to previous studies and were in keeping with lumbar nerve root disease, and
(jj) on 14 March 2025, Dr Coughlan reported that the left side of the applicant's trunk had progressively worsened. Dr Coughlan stated that the applicant had been reviewed by Professor Heard, who pointed out that the three nerve conduction tests had been essentially consistent and pointed towards a nerve root issue in the lumbar spine. Dr Coughlan reported that the applicant had a stable fusion, which had certainly helped his axial back pain. Dr Coughlan reported that the applicant had a synovial cyst at L4/5 which was potentially compressing the L4 nerve within the foramen, and he also had some residual hypertrophic facet joint disease at L5/S 1 which was potentially compressing the L5 nerve root within the foramen. Clinically, Dr Coughlan suspected that the applicant's profound foot drop and progressive foot drop was from the L4 and L5 radicular compression.
Dr Coughlan stated that requires a partial facetectomy to completely decompress the nerve roots within the course of the exit foramen where the compression was, noting that the compression was not so much within the canal, but behind the facet joints and within the neural foramina. Dr Coughlan noted that the CT scan showed some residual facet arthropathy at L4/5 on the left hand side, which was potentially compressing the L4 nerve root in the preforaminal area and foraminal area. Dr Coughlan expressed concern in relation to the ongoing delay, the progression of the applicant's foot drop, the longevity of his foot drop, and the profound impact that was going to have on his gait and his stability and his risk of falls. Given the applicant's ongoing deterioration in terms of nerve function,
Dr Coughlan recommended expediting the decompression.
Dr Caroline Tan, neurosurgeon, independent medical expert qualified by the respondent
(a) reported on her examination of the applicant on 16 December 2024 and review of relevant material including imaging;
(b) recorded a detailed history of injury and subsequent treatment;
(c) recorded that the applicant's current symptoms were constant central low back pain, random spasms in the left foot, the left foot "feels like cardboard" and there is dense numbness in the left shin;
(d) recorded that the applicant's current treatment is physiotherapy and pain medication;
(e) on examination, noted that the applicant walked with a wheelie-walker, had zero power of left ankle dorsiflexion, left foot eversion and of the extensor hallucis longus, however power of left knee flexion and extension was normal, there was bilaterally depressed deep tendon reflexes in both limbs and severe numbness to light touch in the anterolateral left leg;
(f) diagnosed permanent left L5 nerve root injury;
(g) expressed the opinion that "the diagnosis of permanent left L5 nerve injury is attributable to the work injury that occurred on 1 March 2022, although Dr Anil's report does record that the symptoms worsened after the work injury on 1 March 2022, so there may have been subsequent injury after 1 March 2022";
(h) stated that:
"Subject to the qualification that I do not have sufficient information to confirm that his foot drop was complete from 1 March 2022, I believe that his employment and, more specifically, the incident on 1 March 2022 are the cause of the persistent weakness and numbness in the left lower limb, which is entirely consistent with a diagnosis of permanent left L5 nerve root injury. The description of the incident would provide for a mechanism of injury where the left L5 nerve was severely contused and internally disrupted (axonotmesis). If Dr Heard had performed needle EMG, this could have confirmed the diagnosis of left L5 nerve injury and also indicated the poor prognosis for recovery. Mr Bridge stated that the foot weakness was present from the time of the work incident and I am assuming that is true. The gas in the L5/S 1 disc means that the segment was mobile and he could potentially have had further injury to the nerve after the work incident. Furthermore, Mr Bridge and Dr Coghlan have stated that the foot weakness did not alter at all despite the many surgeries he has undergone";
(i) in relation to recommended treatment, stated:
"In terms of the foot weakness, it is my opinion that further spinal surgery is futile. First of all, the original injury could have been of a severity that recovery of the foot weakness was never realistically going to occur. He would have had the best chance of recovery if he had presented to a neurosurgeon immediately upon noticing severe foot drop. However,
Mr Bridge presented late to a neurosurgeon with his foot drop and by then whatever prospects of motor recovery were probably dashed. After nearly three years of complete left foot drop, the very remote possibility that his foot drop will ever recover now could be confirmed by referring him to a neurologist to perform needle EMG. If the needle EMG shows no regenerating axons then it is extremely unlikely that he will experience any recovery of the foot weakness. In that event, the only treatment that I think could be of substantive value, would be functional restoration through nerve or tendon transfers. A plastic surgeon with expertise, skill and knowledge in this area of functional restoration would have to be consulted about whether tendon or nerve transfer would be feasible and worthwhile.
In terms of the back pain, this could be due to his underlying degenerative spinal disease and/or tissue trauma and adjacent segment strain from his fusion surgery. There is no surgery that can reliably relieve such axial pain so he would be best advised to manage this conservatively. An exercise physiologist has already provided him with guidance on how to reduce loading of his lumbar spine but he may also get benefit from a period of exercise sessions with an exercise physiologist. The input of a pain management specialist may be helpful with respect to pharmacological therapies. He may also undergo a pain management program.
Advice about the exact treatment type, frequency and duration will have to be obtained from the treating health practitioners";
(j) expressed the opinion that the treatment proposed by Dr Coughlan was not recommended for the reasons set out above;
(k) expressed the opinion that there would be no benefit from further spinal surgery;
(l) in relation to the treatment proposed by Dr Coughlan, stated that:
"The proposed surgery is not expected to assist with his recovery from any work-related injury, for reasons already given. It would be decompressing the left L4 nerve which is not a cause for severe left foot drop and it would also be treating a pathology that was not present in the earlier postoperative MRIs so the left L4 nerve was not affected by the work incident", and
(m) in relation to alternative treatment, stated that:
"In his current state, if needle EMG confirms axonotmesis or other permanent injury to the left L5 nerve, and given his constant significant back pain, he will not be able to return to suitable employment now or into the future, unless he should undergo successful functional restoration surgery by a plastic surgeon. However, even if he undergoes functional restoration surgery, the timeframe for recovery and full realisation of the benefits of that surgery is in the order of 1 -2 years by which time he will be nearly 60 years age so there is considerable uncertainty about his prospects for return to work."
Dr Peter Spittaler, neurosurgeon, independent medical expert qualified by the applicant
(a) reported on his examination of the applicant on 14 April 2025 and review of relevant material including imaging;
(b) recorded a detailed history of injury and subsequent treatment;
(c) noted that the applicant reported constant lower back pain and left calf pain and persisting weakness in the left foot;
(d) noted that the applicant's current treatment included physiotherapy and pain relief;
(e) on examination, noted that there was evidence of a left L5 radiculopathy with weakness and sensory disturbance, which included that the applicant used a wheelie-walker, there was mild tenderness in the paraspinal muscles, extension of the lumbar spine was limited by left-sided lower back pain, there was a complete left sided foot drop and the left calf measured approximately 1.5 cm less than the right calf, ankle jerks were absent bilaterally, there was reduced sensation in the lateral calf and dorsum of the foot on the left side;
(f) diagnosed lumbar spondylosis, specifically L4/5 and L5/S1 degenerative disc disease and subsequent foraminal stenosis and nerve root compression with a left foot drop;
(g) gave a poor prognosis for relief of the applicant's back pain and left foot weakness and foot drop;
(h) expressed the opinion that the applicant had pre-existing asymptomatic lumbar disc degeneration most severe at L5/S1 but also present at L4/5, which had been exacerbated and rendered symptomatic by the accepted injury in March 2022;
(i) in relation to the treatment proposed by Dr Spittaler, stated:
"On my viewing of the imaging there appears to be some foraminal stenosis laterally from the pathology I have described in the investigations and on that basis I think repeat surgery would be worthwhile. That being said I do not think that a left L4/5 foraminotomy which would decompress the L4 nerve root is likely to have a major impact on the foot weakness although it is possible; weakness of dorsiflexion usually occurs with an L5 nerve root lesion which occurs from L5/S1 foraminal stenosis but this notwithstanding it is possible there is a contribution to the weakness from the L4 nerve root";
(j) in relation to the cause of the need for the treatment, Dr Spittaler expressed the opinion that:
"Either the employment or the treatment for the injury sustained from the employment may be leading to the foraminal stenosis. The foraminal stenosis appears to have developed since the interbody fusion surgery and it is possible there has been a subluxation of the facet joint which has led to the foraminal stenosis developing. It certainly did not appear to be there on the CT scan from March 2024", and
(k) in relation to the report of Dr Tan dated 21 December 2024, Dr Spittaler stated that:
"I do not know that I would describe 'further spinal surgery as futile'. In essence I think it is likely Mr Bridge will have a permanent L5 root lesion. I think there is a possibility that decompressing the L4 root could lead to some improvement although as I have stated above L4 is not usually the major root supply to the tibialis anterior (which is the dorsiflexor of the foot). Nevertheless it often does provide some supply so there may be some improvement in the weakness. There is certainly definitely evidence of foraminal stenosis laterally in the foramen so I can understand why
Dr Coughlan is suggesting the treatment; given the significant neurological deficit I think it is prudent, wise and appropriate to 'pull out all stops' to try and improve the situation. Nevertheless given the length of time and the entrenched changes on electrophysiology I think the likelihood of success is not great but it is possible. In short, if I were Mr Bridge I would want everything done to try and improve the weakness. On that basis I do not think that the surgery should be denied and certainly there is nothing else to do apart from wearing a splint to overcome the weakness. Another point I would make is that the surgery proposed by Dr Coughlan is not a major procedure and is very unlikely to make things worse."
(a) relied on the principles enunciated by Burke CCJ in Bartolo v Western Sydney Area Health Service,[1] that in determining whether a medical treatment will be reasonably necessary, if it is better that he have it, then it is necessary and should not be forborne;
(b) submitted that the medical evidence, in particular that of Dr Spittaler and Dr Tan, demonstrates that the treatment is prudent, wise and appropriate, should not be denied, is unlikely to make things worse;
(c) submitted that no viable alternative treatments have been identified;
(d) submitted that the applicant's statement shows how the applicant used a splint which he found uncomfortable and provided no stability and now he uses a walking stick;
(e) submitted that the proposition the accepted injury did not involve the left L4 nerve (which derives from Dr Tan's report) is not determinative of the issue of whether the treatment is reasonably necessary as a result of the injury;
(f) submitted that Dr Spittaler's opinion is that the treatment or the employment may be leading to the foraminal stenosis, and on that basis, there is a causal link between the employment and worsening of the applicant's symptoms which has been evidenced by the applicant, and
(g) submitted that the treatment is the only reasonably treatment option with any potential effectiveness and it is reasonably necessary as a result of the accepted injury.
(a) submitted that the applicant has not discharged the onus of proof and has not established that the treatment is reasonably necessary as a result of the accepted injury, and the evidence is a wholly undesirable basis to make an order for compensation pursuant to s 60 of the 1987 Act;
(b) submitted that the medical evidence demonstrates that the treatment targets the wrong nerve root, and that the treatment is unlikely to result in any significant improvement in the applicant's condition;
(c) submitted that the pathology targeted by the treatment is unrelated to the accepted injury;
(d) submitted that the evidence does not establish any basis upon which the test in Diab is satisfied because there is consistent evidence that the treatment is not expected to improve the applicant's foot drop which is expected to be permanent, Dr Tan proposed an alternative treatment of tendon or nerve transfers which has not been properly considered, the cost is excessive to engage in an endeavour where there is slim to no chance of effectiveness, the treatment is speculative and is not likely to be effective;
(e) submitted that the treatment is against the applicant's interests, will not benefit him and it may be better for him to explore the alternative treatments proposed by Dr Tan, and
(f) submitted that the Commission should make an award for the respondent.
(a) submitted that the Commission should prefer and accept the evidence of
Dr Spittaler that the accepted injury or employment with the respondent led to foraminal stenosis and that is the basis for the treatment, that there is a chance of a benefit to the applicant of having the treatment, that it is better that the applicant have the treatment than not have the treatment, that the characterisation of the applicant's condition as permanent is not supported by the evidence, and that the respondent has not adduce evidence that the alternative treatments proposed by Dr Tan are appropriate and anything more than options to be considered.
"60 Compensation for cost of medical or hospital treatment and rehabilitation etc
(1) If, as a result of an injury received by a worker, it is reasonably necessary that:
(a) any medical or related treatment (other than domestic assistance) be given, or
(b) any hospital treatment be given, or
(c) any ambulance service be provided, or
(d) any workplace rehabilitation service be provided,
the worker's employer is liable to pay, in addition to any other compensation under this Act, the cost of that treatment or service and the related travel expenses specified in subsection (2)."
"...Treatment is necessarily purposive. Treatment, in the medical or therapeutic context, relates to the management of disease, illness or injury by the provision of medication, surgery or other medical service designed to arrest or abate the progress of the condition or to alleviate, cure or remedy the condition. It is the provision of such services for the purpose of limiting the deleterious effects of a condition and restoring health. If the particular 'treatment' cannot, in reason, be found to have that purpose or be competent to achieve that purpose, then it is certainly not reasonable treatment of the condition and is really not treatment at all. In that sense, an employer can only be liable for the cost of reasonable treatment..." (my emphasis)
"The standard test adopted in determining if medical treatment is reasonably necessary as a result of a work injury is that stated by Burke CCJ in Rose v Health Commission (NSW) [1986] NSWCC2; [1986] NSWCC 2; (1986) 2 NSWCCR 32 (Rose) where his Honour said, at 48A-C:
"The question is should the patient have this treatment or not. If it is better that he have it, then it is necessary and should not be forborne. If in reason it should be said that the patient should not do without this treatment, then it satisfies the test of being reasonably necessary."
"In the context of s 60 the relevant matters, according to the criteria of reasonableness, include, but are not necessarily limited to, the matters noted by Burke CCJ at point (5) in Rose (see [76] above), namely:
(b) the appropriateness of the particular treatment;
(c) the availability of alternative treatment, and its potential effectiveness;
(d) the cost of the treatment;
(e) the actual or potential effectiveness of the treatment, and
(f) the acceptance by medical experts of the treatment as being appropriate and likely to be effective.
With respect to point (d), it should be noted that while the effectiveness of the treatment is relevant to whether the treatment was reasonably necessary, it is certainly not determinative. The evidence may show that the same outcome could be achieved by a different treatment, but at a much lower cost. Similarly, bearing in mind that all treatment, especially surgery, carries a risk of a less than ideal result, a poor outcome does not necessarily mean that the treatment was not reasonably necessary. As always, each case will depend on its facts."
Is the treatment reasonably necessary as a result of injury?
"From the earliest days of compensation legislation, it has been recognised that causation is not always direct and immediate...
Since that time, it has been well recognised in this jurisdiction that an injury can set in train a series of events. If the chain is unbroken and provides the relevant causative explanation of the incapacity or death from which the claim comes, it will be open to the Compensation Court to award compensation under the Act."[7]
"The result of the cases is that each case where causation is in issue in a workers' compensation claim, must be determined on its own facts. Whether death or incapacity results from a relevant work injury is a question of fact. The importation of notions of proximate cause by the use of the phrase 'results from', is not now accepted. By the same token, the mere proof that certain events occurred which predisposed a worker to subsequent injury or death, will not, of itself, be sufficient to establish that such incapacity or death 'results from' a work injury. What is required is a commonsense evaluation of the causal chain. As the early cases demonstrate, the mere passage of time between a work incident and subsequent incapacity or death, is not determinative of the entitlement to compensation. In each case, the question whether the incapacity or death 'results from' the impugned work injury (or in the event of a disease, the relevant aggravation of the disease), is a question of fact to be determined on the basis of the evidence, including, where applicable, expert opinions. Applying the second principle which Hart and Honoré identify, a point will sometimes be reached where the link in the chain of causation becomes so attenuated that, for legal purposes, it will be held that the causative connection has been snapped. This may be explained in terms of the happening of a novus actus. Or it may be explained in terms of want of sufficient connection. But in each case, the judge deciding the matter, will do well to return, as McHugh JA advised, to the statutory formula and to ask the question whether the disputed incapacity or death 'resulted from' the work injury which is impugned."
"57. The work injury does not have to be the only, or even a substantial, cause of the need for the relevant treatment before the cost of that treatment is recoverable under s 60 of the 1987 Act.
"It is therefore not necessary for Mr Moon to establish that he suffered an 'injury' to his left shoulder within the meaning of that term in section 4 of the 1987 Act. All he has to establish is that the symptoms and restrictions in his left shoulder have resulted from his right shoulder injury. Therefore, to the extent that the Arbitrator and Dr Huntsdale approached the matter on the basis that Mr Moon had to establish that he sustained an 'injury' to his left shoulder in the course of his employment with Conmah they asked the wrong question."
"The Commission has considered and explained the difference between an 'injury' and a condition that has resulted from an injury in several recent decisions...
The injury to Mr Bouchmouni's right knee caused him to seek treatment in the form of surgery and physiotherapy. The evidence suggests that it was in the course of receiving that treatment, and/or as a result of an altered gait because of his knee symptoms, Mr Bouchmouni developed back symptoms. If that is accepted, and no reason has been advanced why it should not be, it is clear beyond doubt that his back condition has resulted from the treatment he received for his accepted knee injury and his altered gait. That does not, however, make the back condition an 'injury'."
"81. It follows that the description of a personal injury as 'a sudden identifiable pathological change' is consistent with the authorities. It suggests no more than that, to qualify as a personal injury, there must be some sudden and ascertainable or dramatic physiological change or disturbance of the normal physiological state. Such a change or disturbance may be as simple as a bruise or a soft tissue strain. If the personal injury also aggravates a pre-existing disease, that does not mean it is no longer a personal injury."
"(1) A finding that a fact exists (or existed) requires that the evidence induce, in the mind of the fact-finder, an actual persuasion that the fact does (or at the relevant time did) exist;
(2) Where on the whole of the evidence such a feeling of actual persuasion is induced, so that the fact-finder finds that the probabilities of the fact's existence are greater than the possibilities of its non-existence, the burden of proof on the balance of probabilities may be satisfied;
(3) Where circumstantial evidence is relied upon, it is not in general necessary that all reasonably hypotheses consistent with the non-existence of a fact, or inconsistent with its existence, be excluded before the fact can be found, and
(4) A rational choice between competing hypotheses, informed by a sense of actual persuasion in favour of the choice made, will support a finding, on the balance of probabilities, as to the existence of the fact in issue."
Dr Coughlan reported that the applicant had a synovial cyst at L4/5 which was potentially compressing the L4 nerve within the foramen, and he also had some residual hypertrophic facet joint disease at L5/S 1 which was potentially compressing the L5 nerve root within the foramen. Clinically, Dr Coughlan suspected that the applicant's profound foot drop and progressive foot drop was from the L4 and L5 radicular compression. Dr Coughlan stated that requires a partial facetectomy to completely decompress the nerve roots within the course of the exit foramen where the compression was, noting that the compression was not so much within the canal, but behind the facet joints and within the neural foramina.
Dr Coughlan noted that the CT scan showed some residual facet arthropathy at L4/5 on the left hand side, which was potentially compressing the L4 nerve root in the preforaminal area and foraminal area. Dr Coughlan expressed concern in relation to the ongoing delay, the progression of the applicant's foot drop, the longevity of his foot drop, and the profound impact that was going to have on his gait and his stability and his risk of falls. Given the applicant's ongoing deterioration in terms of nerve function, Dr Coughlan recommended expediting the decompression. 7. I am satisfied that the above evidence demonstrates that the applicant has ongoing symptoms which included pain, but is primarily left-sided foot drop. The applicant's treating neurosurgeon, Dr Coughlan, has explained that recent MRI scan showed a synovial cyst at L4/5 which is compressing the left L4 nerve root and opines that such radicular pathology could be the cause of the applicant's ongoing symptoms and that the treatment may provide relief.
The availability of alternative treatment, and its potential effectiveness
The acceptance by medical experts of the treatment as being appropriate and likely to be effective
Dr Tan expressed the opinion that the treatment would be futile to provide any relief to the applicant because:
(a) the original injury could have been of a severity that recovery of the foot weakness was never realistically going to occur;
(b) the applicant presented late to a neurosurgeon with his foot drop and by then whatever prospects of motor recovery were probably dashed, and
(c) the treatment would be decompressing the left L4 nerve which she did not believe was a cause of the applicant's severe left foot drop.
Is the treatment reasonably necessary as a result of the injury?
(a) the stereotactic guided left L4/5 far lateral foraminotomy requested by
Dr Coughlan is reasonably necessary treatment as a result of the injuries, pursuant to s 60 of the 1987 Act.
(a) in accordance with s 60 of the 1987 Act_,_ the respondent to pay the past costs of and incidental to stereotactic guided left L4/5 far lateral foraminotomy requested by Dr Coughlan.
[2] [1986] NSWCC2; [1986] NSWCC 2; (1986) 2 NSWCCR 32.
[4] [1986] NSWCC2; [1986] NSWCC 2; (1986) 2 NSWCCR 32.
[7] Kooragang, at [461] (Sheller and Powell JJA agreeing).
[8] [2016] HCA 43, at [42].
[11] See also Kumar v Royal Comfort Bedding Pty Ltd [2012] NSWWCCPD 8, at [61].
Bridge
J Hutchinson Pty Ltd [2025] NSWPIC 639;
(1994) 35 NSWLR 452
(1986) 2 NSWCCR 32
(1996) 12 NSWCCR 716