(d) Does a hemiparesis, caused by a stroke and with no active movement of the ankle or foot, impact upon the assessment of impairment of the affected ankle and foot? If yes, in what way does a stroke impact upon the assessment of impairment of the affected ankle and foot?
Yes. If one is assessing impairment of the ankle and hindfoot based on the AMA Fourth Edition Guidelines and using the range of motion model, then one is dependent on a patient having the ability to actively move the ankle and hindfoot joints. If they have had a stroke that has led to musculoskeletal weakness, spasticity, deformity etc, then assessing impairment of an injury separate to a stroke would be extraordinarily difficult using the range of motion model.
(e) Is the assessment of impairment of the foot and ankle undertaken pursuant to section 3.2e at page 77 of [the Guides], being the range of motion method, significantly affected by a hemiparesis where there is no active movement of the foot or ankle consequent to a stroke?
Yes. As outlined above.
(f) Is the assessment of impairment of an ankle and foot, in a person with a hemiparesis who has no active movement of the foot or ankle consequent to a stroke, most appropriately undertaken by applying:
(i) section 3.2i, the Diagnosis-Based Estimates assessment method at page 84, and Table 64 of the Guides? or
(ii) section 3.2e, the range of motion method, at page 77 of the Guides? or
(iii) section 3.2f, the joint ankyloses method, at page 79 of the Guides? or
(iv) other, and if so what, section of the Guides?
This is a very difficult question to answer. If one takes the type of ankle fracture that [the defendant] sustained, i.e. a standard closed Weber C fracture, then one would have expected following recovery for him to have had some mild restriction of range of motion, i.e. dorsiflexion and plantar flexion of the ankle joints. One would have expected him to have noted in time some intermittent ankle pain in relation to impact physical activity, prolonged walking and standing etc. The observation or development of flattening of the foot and valgus deformity at the midfoot three months or so post injury is very unusual. Very rarely in association with an ankle fracture, a rupture of the tibialis posterior tendon can occur. If this occurred and was not diagnosed, then in ·time planovalgus, i.e. valgus and flat foot deformity can occur. Investigation of [the defendant] showed that tibialis posterior tendon was intact. Minor tendinopathy at its insertion would be extraordinarily common in a patient of his age. A stroke resulting in a significant hemiparesis throws a large spanner in the works in relation to assessment of range of motion of the ankle joint and hindfoot. In time, in relation to a stroke consequent upon weakness and spasticity, fixed deformity can occur. There is no satisfactory classification in the Guides to allow assessment of impairment in this regard.
(g) In the absence of joint ankylosis, is the application of section 3.2f, the joint ankyloses method, at page 79 of the Guides appropriate in determining impairment?
In my view, [the defendant] did not develop ankylosis of the ankle joint, hindfoot etc consequent upon his ankle injury. I do not believe that one could use ankylosis as a satisfactory measurement of impairment etc in [the defendant's] case.