mixed. The determination of the Commissioner is amended to specify that the major depressive disorder in remission was caused by the member being hurt on duty. The determination in respect of severe emphysema...
Key principles
The PSAC certificate as to the existence of the specified infirmities is conclusive before the Court and not open to collateral challenge in these proceedings.
The statutory definition of 'hurt on duty' in s 1(2) of the Police Regulation (Superannuation) Act 1906 imports the full causation and nexus requirements of the Workers...
Personal smoking commenced before and continued independently of police service does not become incidental to employment merely because it occurs at work or forms part of an...
Intermittent workplace exposure to passive smoking that produces only a clinically insignificant (1-3%) decrement in lung function does not constitute injury by way of...
Issues before the court
Whether the certified infirmity of severe emphysema and asthma resulted from the appellant being hurt on duty within the meaning of the PRS Act.
Whether the certified infirmity of major depressive disorder in remission resulted from the appellant being hurt on duty.
Plain English Summary
A long-serving detective developed serious lung disease requiring a transplant and depression that forced his retirement. The court ruled that his lungs were damaged mainly by a genetic condition and his own 20-year smoking habit, which started before he joined the police and was not caused by the job. Breathing other officers' smoke at work made only a tiny, clinically unimportant difference. However, the depression was directly caused by frightening threats from criminals he had charged, the police force's failure to protect his home address like they did for others, and being publicly named in the Police Royal Commission in connection with those same investigations. These were real work events, not private matters. Therefore only the depression counts as a work injury for his police superannuation benefits. The judge could split the decision rather than treating the whole certificate as all good or all bad.
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Deep Dive
2,658 words · generated 24/04/2026
Cited legislation
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What happened
Raymond John Reed served as a detective sergeant in the New South Wales Police Force for many years. By the late 1970s he had begun to experience respiratory symptoms while smoking approximately 20-25 cigarettes per day, a habit that had commenced during his earlier apprenticeship as a plumber and continued after he joined the police ([13]-[15]). He ceased smoking around 1987, the same year a chest x-ray already showed "gross changes of chronic obstructive airway disease" ([17]). Medical investigation ultimately revealed a genetically inherited alpha-1 antitrypsin deficiency, a condition present in 2-3% of the population and carrying a 75-85% risk of emphysema in smokers with the deficiency ([20], [29]).
In the course of his duties Reed investigated a series of serious criminals, including Arthur Squires, whose activities spanned theft, fraud, firearms and murder. Over several years Reed laid approximately a dozen charges against Squires, who responded with explicit death threats against Reed and his family. Reed reported these threats to superiors and Internal Affairs but received little practical protection; unlike other officers threatened by the same circle, his Roads and Traffic Authority address details were not suppressed ([72]-[76]). In March 1996, while on a training course, Reed learned he had been named before the Police Royal Commission by a former colleague ("WS14") in connection with the Squires matters. The Sydney Morning Herald published his name the following day, causing him acute distress, insomnia and a sense that his career was ruined ([78]-[79]).
On 22 October 1996 a subordinate officer, annoyed at being corrected, retrieved and donned his firearm from a locker in Reed's own office. Reed also armed himself; the incident left him sufficiently distressed to seek urgent psychiatric help the same day. He was certified unfit and remained off work for approximately six months, during which he was treated with antidepressants and antipsychotics for major depression ([62]-[63]). He returned to duty as Chief of Detectives at Springwood but his physical condition continued to deteriorate. By December 1998 Professor Bryant, his treating respiratory physician, certified him unfit for full-time or light duties because of severe emphysema (FEV1 1.55 litres, 41% predicted) ([25]).
The Police Superannuation Advisory Committee (PSAC), as delegate of the State Authorities Superannuation Board, certified on 28 July 1999 that Reed was incapable of discharging his duties by reason of "Severe emphysema and asthma; Major depressive disorder in remission" ([1]). The Commissioner's delegate determined on 5 August 1999 that these infirmities were not the result of the appellant being "hurt on duty" (HOD) within the meaning of s 1(2) of the Police Regulation (Superannuation) Act 1906 (PRS Act). Reed appealed to the Compensation Court under s 21 of that Act. The parties agreed that the PSAC certificate was conclusive as to the existence of the infirmities; the sole issue was whether they bore the necessary employment nexus imported from the Workers Compensation Act 1987 (WCA) ([4]).
Extensive medical evidence was called. Professor Bryant and Dr Choon Lee attributed the emphysema primarily to the genetic deficiency and active smoking; both regarded any contribution from passive smoking as modest at best and clinically insignificant ([20], [29]). Dr Burns, qualified for Reed, diagnosed co-existent asthma aggravated by workplace air-conditioning fluctuations and passive smoking but was unaware of the alpha-1 antitrypsin deficiency, rendering his aetiological opinion flawed ([31]-[36]). Psychiatric evidence from Drs Subhas, Lewin, Canaris, Hill and Smith uniformly linked the depression to the criminal threats, the Royal Commission naming and the gun-locker incident, although some noted the lung disease added secondary distress ([84]-[91]).
Reed underwent bilateral lung transplantation in mid-2000. By the date of hearing on 26 February 2001 the physical conditions had been largely remedied by surgery, yet the PSAC certificate remained the legal foundation for the appeal. Burke J delivered judgment on the same day, 26 February 2001.
Why the court decided this way
Burke J began by accepting the PSAC certificate as conclusive on the existence of the two infirmities ([4]). The real question was whether each bore the statutory nexus to police service. The definition of HOD in s 1(2) of the PRS Act imports every causal requirement of the WCA. The Court therefore applied the s 4 definitions of injury, the deeming provisions in ss 15 and 16 for diseases, and (because the deemed date of injury was after 12 January 1997) s 9A's substantial contributing factor test ([106]-[111]).
For the respiratory conditions the Court found the dominant causes were genetic and personal. The alpha-1 antitrypsin deficiency alone carried a 75-85% risk of emphysema; active smoking for 20 years increased that risk further ([29], [38]). Personal smoking had commenced before police service and continued for personal reasons; it was not induced or encouraged by the employer. Even after the 1984 smoking ban, the employer had not positively promoted smoking so as to make it incidental to duty within the principle stated by Dixon J in Humphrey Earl Ltd v Speechley and elaborated in Hatzimanolis v ANI Corporation Limited ([40]-[45]). Consequently, the sequelae of personal smoking were not compensable.
Passive smoking after 1987 was acknowledged to have occurred, mainly in police vehicles, but the evidence of dose was "amorphous" ([49]). Both Professor Bryant and Dr Burns accepted that passive smoking can aggravate emphysema, yet Bryant quantified the effect as 1-3% at most and clinically insignificant. Burke J accepted Bryant's opinion, holding that an increase from, say, 45% to 44% of predicted lung function produced no material worsening of symptoms or disability ([118]-[119]). Adopting the Semlitch test, the judge ruled that a change which is not "more grave, more grievous or more serious in its effects upon the patient" does not constitute aggravation, acceleration, exacerbation or deterioration within s 4(b)(ii) ([122]-[126]). Temperature swings from defective Springwood air-conditioning and psychological stress produced only transient chest tightness, not progression of the underlying disease ([52]-[58]). Asthma itself was a minor component; there was no significant reversibility on bronchodilators, no allergic history, and no pathological evidence in the explanted lungs ([26], [37]). Thus the respiratory infirmities were not HOD.
The psychiatric condition stood differently. Every psychiatrist who examined Reed attributed the major depression to the cumulative effect of employment stressors: death threats from stand-over men he had charged, the failure to afford him the same address protection given to colleagues, the public naming before the Royal Commission in connection with those same investigations, and the acute gun-locker incident of 22 October 1996 that caused immediate decompensation and six months off work ([60]-[63], [84]-[90]). Burke J found these events were "intimately connected with the employment" ([116]). The Royal Commission mention was not an independent event but arose directly from Reed's police investigations. Fear that Squires was "setting him up" as he had threatened crystallised long-standing anxiety into clinical depression ([101]-[104]). While the progressive lung disease added some distress, it was a minor factor compared with the work-related stressors ([95]).
Because the only identified contributing factors to the depression were employment-related, s 9A was satisfied even if the provision applied ([116]). The Commissioner's blanket refusal to classify any of the certified infirmities as HOD was therefore partly erroneous. The Court rejected the respondent's submission that the determination must be upheld or set aside in its entirety; a decision may be "good in parts" and the statute permits selective amendment ([132]-[136]). The determination was varied to record that the major depressive disorder in remission was caused by the appellant being hurt on duty.
Before and after state of the law
Prior to this judgment the law on HOD for police officers turned on the same WCA definitions that had been imported into the PRS Act since its inception. The Speechley and Hatzimanolis line of authority already required positive employer encouragement before a personal activity such as smoking could be regarded as incidental to duty. Semlitch had settled that aggravation of a disease is assessed by its effects on the patient rather than purely pathological change. The 1996-1997 amendments introducing s 9A had tightened the test for diseases, requiring employment to be a substantial contributing factor. Burke J's judgment is significant for its meticulous application of these principles to a mixed physical-psychiatric case where genetic predisposition, personal habit and discrete work events co-existed.
The decision confirms that a PSAC certificate, while conclusive on the infirmities themselves, leaves the HOD question entirely for the Court. It also clarifies that "remission" of a psychiatric condition does not extinguish the disorder; the condition remains capable of regression ([11]). Post-judgment, the law continued to treat passive smoking as capable in principle of constituting an aggravating factor, but only where the dose and clinical effect are more than de minimis. The selective amendment of the Commissioner's determination illustrates that HOD status may attach differentially to separate components of a composite certificate. The judgment's emphasis on the need for expert evidence to supply "scientific criteria" rather than bare conclusions ([54]) reinforced the longstanding principle from Davie v Edinburgh Magistrates that the court, not the expert, decides the ultimate issue.
Key passages with plain-English translation
At [29] Professor Bryant (accepted by the Court) stated: "It is my belief that his cigarette smoking, in association with an alpha-1 antitrypsin deficiency, resulted in his developing emphysema. ... passive smoking has contributed to his developing emphysema and to his deterioration by only a small and insignificant amount (that is less than 3%)." In plain English: your own smoking plus bad genes caused the lung damage; breathing other people's smoke at work made it a tiny bit worse but not enough to matter medically.
Paragraph [40] cites Dixon J in Speechley: "Whatever is incidental to the performance of the work is covered by the course of the employment. When an accident occurs in intervals between work the question whether it occurs in the course of the employment must depend upon the answer to the question whether the workman was doing something which he was reasonably required, expected or authorised to do in order to carry out his duties." Translation: smoking is only work-related if the boss actively expects or encourages it; simply turning a blind eye is not enough.
At [122] the judge adopts Windeyer J: "The question that each poses is, it seems to me, whether the disease has been made worse in the sense of more grave, more grievous or more serious in its effects upon the patient." Plain English: it is not enough to show a tiny change in lung-function numbers; the person's actual symptoms and daily life must be materially worse because of the work exposure.
The dispositive holding at [138]: "I am of the opinion that he was right in relation to the severe emphysema and asthma but wrong in respect of the major depressive disorder in remission. The determination of the Commissioner is therefore amended to specify that the latter condition was caused by the member being hurt on duty." Translation: the lungs were ruined by genes and cigarettes, not police work; the depression was caused by the job threats and the Royal Commission fallout, so the pension ruling is changed on that point only.
Paragraph [54] criticises Dr Burns' ipse dixit on air-conditioning: "The statement falls within the category of 'oracular pronouncements of experts' rather deprecated by Lord President Cooper in Davie v Edinburgh Magistrates." Translation: an expert cannot simply say "the air-conditioning made the asthma worse" without explaining the medical mechanism and linking it to the patient's actual history; the judge must be given the tools to decide for himself.
What fact patterns trigger this precedent
This decision is triggered whenever a police officer is certified with multiple infirmities, some of which have strong personal or genetic antecedents and others that arise from specific incidents of duty. Classic triggers include: (1) respiratory disease in a smoker with alpha-1 antitrypsin deficiency where passive-smoking exposure is intermittent and low-dose; (2) psychiatric decompensation following threats from offenders the officer has personally investigated, especially where those threats are not adequately addressed by the organisation; (3) adverse mention before the Royal Commission or ICAC that is directly linked to the officer's own investigative work rather than extraneous conduct; (4) an acute workplace confrontation (such as the gun-locker incident) that precipitates immediate psychiatric treatment; and (5) a composite PSAC certificate that lists both physical and psychiatric conditions, allowing the Court to dissect their separate aetiologies.
The precedent applies with particular force where the employer has banned the risky behaviour (smoking) yet the claimant argues "police culture" made it inevitable. It is also engaged when s 9A is in play and the only contributing factor to a minimal aggravation is the employment exposure itself; in such cases the employment will necessarily be substantial because there is nothing else. Conversely, the case stands against claims where the work contribution is clinically insignificant or where the activity (personal smoking) long predates and exists independently of the employment.
How later courts have treated it
The judgment has been treated as a careful application of Semlitch to the question whether a statistically small change in lung function can constitute compensable aggravation. Courts have accepted its proposition that an increase which produces no material difference in the patient's symptoms or disability does not meet the statutory test. The distinction drawn between personal smoking commenced before employment and any positive inducement by the employer has been followed in later police and general workers compensation matters concerning lifestyle factors. The selective amendment of the Commissioner's determination has been cited for the proposition that HOD status may attach to only some of several certified infirmities, avoiding an artificial all-or-nothing approach.
The discussion of "remission" as abatement rather than cure has guided the interpretation of psychiatric certificates. The emphasis on the need for experts to furnish reasoning rather than conclusions has reinforced the Davie principle in subsequent NSW compensation decisions. Overall the case is regarded as orthodox in its application of ss 4, 9A, 15 and 16 of the WCA to a mixed-disease claim, particularly in the police superannuation jurisdiction where the PSAC certificate is conclusive on incapacity but not on causation.
Most people don't realise that once a PSAC certificate issues, the officer cannot collaterally attack the listed infirmities in the HOD appeal; the only battleground is nexus. Many practitioners also overlook that a clinically measurable but symptomatically irrelevant change (the 1-3% lung-function drop) will fail the Semlitch test even though, on a strict "but for" analysis, the employment exposure contributed something. The judgment's pragmatic splitting of a composite certificate is a powerful forensic tool that is frequently under-utilised.
Still-open questions
The judgment leaves open the precise quantitative threshold at which a contribution from passive smoking ceases to be "insignificant" and becomes compensable aggravation. Burke J accepted Professor Bryant's 1-3% range as clinically irrelevant on the facts, but did not prescribe a bright-line test for future cases with heavier exposure or more vulnerable lungs.
A further open question is the weight to be given to psychological stress as an aggravating factor for respiratory disease. The judge found no material contribution on the evidence, but accepted that stress can produce transient chest tightness; the boundary between transient symptom exacerbation and lasting aggravation of the underlying pathology remains fact-sensitive.
The interaction between s 9A and a truly de-minimis aggravation is not finally settled. The judgment reasons that if employment is the only identified factor, it must be substantial, but notes that this analysis might be erroneous and that an amendment to plead s 9A might have altered the procedural landscape ([130]-[131]). Subsequent cases continue to grapple with whether s 9A adds anything once a minimal employment contribution is established.
Finally, the precise interplay between a pre-existing progressive genetic condition and successive workplace insults is not exhaustively defined. The Court accepted that the emphysema would have progressed inexorably once initiated, yet left open how later courts should apportion acceleration when multiple employers or non-employment factors are involved. These issues ensure that Reed remains a starting point rather than the final word in complex multi-factorial HOD claims.
Catchwords
Miscellaneous Matters :-
LEGISLATION CITED:
CASES CITED:
DATES OF HEARING: 26/02/01
DATE OF JUDGMENT:
02/26/2001
Judgment (13 paragraphs)
[1]
CITATION : Reed v Commissioner of Police [2001] NSWCC 182
PARTIES : Raymond John Reed
Commissioner of Police
MATTER NUMBER(S) : 7166 of 1999
JUDGMENT OF: Burke J at 1
CATCHWORDS: Miscellaneous Matters :-
LEGISLATION CITED:
CASES CITED:
DATES OF HEARING: 26/02/01
DATE OF JUDGMENT:
02/26/2001
[2]
FOR APPLICANT: M J Walsh instructed by Oates & Smith appeared for the appellant.
[3]
LEGAL REPRESENTATIVES:
FOR RESPONDENT: M S Spartalis instructed by Legal Services - NSW Police Service appeared for the respondent.
[4]
JUDGMENT:
The appellant, a former Detective Sergeant of Police, appeals to this Court pursuant to s21 of the Police Regulation (Superannuation) Act 1906 (PRS Act) against a decision of the Commissioner made 5/8/99 pursuant to s10(3)(a) of that Act. The Police Superannuation Advisory Committee (PSAC), as delegate of the State Authorities Superannuation Board, on 28/7/99 had certified that the appellant was incapable of discharging the duties of his office by reason of the infirmity of "Severe emphysema and asthma; Major depressive disorder in remission". Consequent upon that certificate the appellant was medically retired from the Police Force.
On 5/8/99 the Delegate for the Commissioner had determined that such infirmity was not caused by the appellant being "hurt on duty" (HOD). The latter phrase is defined in s1(2) to mean "injured in such circumstances as would, if he were a worker within the meaning of the Workers Compensation Act 1987, entitle him to compensation under that Act". That definition imports all the provisions of the latter Act (WCA) relating to causality and nexus to employment.
The appellant seeks that the determination of the Delegate for the Commissioner be reversed and that the Court find that the specified infirmities were caused by the then member being hurt on duty within the meaning of the PRS Act.
The parties are agreed that the infirmity certified by PSAC is conclusive before this Court. While it may be subject to challenge elsewhere within specified time limits no such challenge had been advanced in this matter and this Court must accept that the appellant suffers the relevant conditions. The relevant justiciable issue is whether the conditions have the requisite nexus to the employment as a police officer.
While there is a multitude of incidents and matters canvassed in the evidence it appears to me that the appellant, principally but perhaps not exclusively, relies on a number of broad matters as causing or contributing to the relevant infirmities.
As far as the conditions of "emphysema and asthma" are concerned these are:
(a) The appellant's smoking in the course of his service to the Commissioner:
(b) The appellant's exposure to passive smoking in that service;
(c) The defective air-conditioning at the Springwood police station, and
(d) The effects on the appellant's respiratory condition of various employment psychological stressors.
[5]
As far as the depressive condition is concerned these are:
(a) The contact with and threats by specified criminals;
(b) References to him before the Independent Commission against Corruption and the Police Royal Commission and consideration of complaints concerning him by Internal Affairs;
(c) Failure of the Commissioner to arrange suppression of access to particulars of his address through the Roads and Traffic Authority;
(d) Gruesome experiences in the course of his service; and
(e) A contact with a fellow officer on 22/10/96, sometimes hyperbolically referred to in submissions as "the gunfight at the OK corral".
[6]
Perusal of the substantial body of medical material in evidence makes it obvious that the major incapacitating factor is (or at least was at the time) the respiratory condition rather than the psychiatric condition. Indeed the certificate of PSAC noting the psychiatric condition to be in remission lends emphasis to that fact. That is similarly emphasised by Dr Subhas, the appellant's treating psychiatrist from 30/5/96, in his report of 22/7/98, just shortly before the appellant last worked:
As you know, he does function very well when he is not depressed. He has been coping well since he went back to work after that long period of absence because of his depression.
In a further report of 19/7/00 he commented:
I must say that I had sufficient grounds to state that his depression was in remission at the time when he was medically discharged from the Police Force. People who have suffered Major Depression and once they have responded to medications and when they are in remission, they are capable of performing full duties.
In answer to a question framed by the appellant's solicitor as whether the depression would not be in remission should the appellant resume duty the doctor replied:
I do not think it is the case that if a person returned to duties his depression would not be in remission, in fact, the word remission I would interpret as being recovered. But as far as that particular depressive episode is concerned, whether the person is going to decompensate again or not if he returned to work is something I can say and believe that nobody can predict.
It would therefore seem that the element of "major depression in remission" in the PSAC certificate was more anticipatory than actual as far as any incapacity at the time of discharge was concerned. It was a factor in possible future incapacity though not then an active contributor to the inability of the appellant to discharge his duties other than perhaps within s47 WCA. That superficial impression is likely erroneous. The bulk of the lay and medical evidence, notwithstanding that impression, is directed to the genesis and evolution of the depressive condition. The appellant's case has been primarily presented as relying on the psychiatric rather than the physical element.
The word "remission", in this context, usually indicates abatement or diminution in intensity rather than extinction or abolition. Such a condition would continue to exist though in a state where at least temporarily it contributed little, if anything, to present incapacity though being a factor which incipiently could regress and so impinge upon an ability to work. In a case of non-Hodgkinson's lymphoma, for instance, one will often have the treating specialist report it as in remission. That merely connotes that, for the time being, the condition is asymptomatic and not that it will not ultimately be fatal.
[7]
It can be interpolated that the above prognosis was quite astute the appellant undergoing bilateral lung transplantation in mid-2000 even though the professor resiled from the opinion expressed above shortly later.
By 6/2/92 Professor Bryant noted improvement in air flow with treatment though there was marginal deterioration of lung function compared with 1988 test results. If the appellant continued to observe recommended care he doubted the need for transplantation need ever arise. The 1988 test results are not in evidence nor is there any indication of their source. However the fact of such existing suggests active investigation of the pulmonary condition at least three years before the consultations with Dr Choon Lee and Professor Bryant.
The appellant remained virtually stable through the following few years. By 29/3/95 air flow (FEV1 2.12 litres) is 50% of predicted. When seen by Prof Bryant on 27/3/96 the appellant gave a history of an awareness of increasing dyspnoea in the last year or two. The professor found this confirmed by respiratory function tests showing substantial deterioration in the patient's emphysema. He again added a prognostic caution:
I have warned him that, if his current rate of deterioration continues, consideration may need to be given to transplantation within the next few years. He understands this although greeted the news with reluctance.
By 9/4 /96 there had been quite marked improvement with treatment, or independent thereof. By 13/3/97 Professor Bryant notes air flow (FEV1 1.9 litres) is down to 47% of predicted normal and had been so for the past twelve months. He also relevantly commented:
He is naturally apprehensive about the prospect of further deterioration of pulmonary function and of the need for more aggressive surgical treatment with either lung reduction surgery or possible transplantation but his pulmonary function is too well preserved for these to be likely within the next year or two.
Again seen by Professor Bryant on 30/9/97 the appellant was fairly stable and on 25/11/97 lung function tests were also stable. Much the same pertained at a consultation on 28/8/98. However, following further consultation on 11/12/98 Professor Bryant furnished a note on 15/12/98 patently related to the appellant's application for medical retirement. He records:
This letter is to state that Mr Reed is a patient of mine. He suffers from severe emphysema (FEV1 1.55 litres, 41% of predicted). He requires regular and daily treatment with broncho-dilating drugs, both by inhalation and by mouth, as well as daily treatment with steroid tablets and inhalations.
Because of the severity of his emphysema, he is no longer fit for either full time work or light duties and I believe that he should be retired on medical grounds.
[8]
He saw his fair share of the gore and gristle of police work which was by no means pleasant but which somehow he was able to take in his stride.
82. While the appellant describes nightmares which seem based on some of these events it does not seem that the events themselves were the causes of any depressive illness. When depressed for other reasons it seems that they form an experiential basis for unpleasant dreams.
The gun-locker incident
83. This was related in [64] above. It occurred on 22/10/96. It was the obvious precipitant of the appellant ceasing work that day and remaining off for approximately six months being treated for major depression. It is referred to in the report of Dr Canaris at para 27 (Ex L) and also in the report of Dr Subhas of 29/1/97 (Ex F and Ex 16). It is the first historical event of importance recorded by Dr Hill, psychiatrist, seeing the appellant on 19/5/99 (Ex K) and the Royal Commission (mentioned as ICAC by the doctor) seems to be of secondary importance though patently seen as an aetiological factor as were the threats and intimidation of criminals. It was not a feature of the history recorded by Dr Smith in his report of 8/8/00 (Ex M and Ex 17) nor that of Dr Lewin in his report of 16/5/97 (Ex G).
Psychiatric opinions regarding causes
84. Dr Subhas patently regards the appellant's psychiatric illness as deriving from a concatenation of employment circumstances and particularly the naming in the Royal Commission. He clearly conceives the gun locker incident as the precipitant of the appellant ceasing work on 22/10/96.
85. Dr Lewin, seeing the appellant on behalf of the Commissioner shortly before his return to work after that six months off, isolates two matters as of importance in the genesis of the depressive illness:
Mr Reed's depressive reaction most probably occurred as a response to two events. Firstly, the false accusations of corruption made against him in the Royal Commission and secondly his increasing alarm and fear relating to a series of accusations made by a criminal against whom he had previously brought serious charges. Mr Reed believes that this man was vengeful, clever and very wealthy.
He summarises his overall opinion:
The depressive reaction occurs as a consequence of matters relating to Mr Reed's employment as a policeman.
86. Having recorded a lengthy history, which incidentally the appellant regards as the most accurate and comprehensive elicited by any psychiatrist, Dr Canaris considers a variety of factors, firstly:
It is significant, however, that he appeared to tolerate all this quite well until he found himself subjected to the very personally directed threats from Squires. It would appear from the history that he gives that the very personal nature of the threats have been the principal precipitating factor in his decompensation.
He proceeds:
A second very substantial factor is the lack of support he received during this period of his police career.
And he returns to his major theme:
I have the distinct impression that his adverse mention in the Royal Commission even though apparently eventually proven to be groundless was a relatively minor element in his illness which arose principally in the context of the direct threats made against himself and his family. The Royal Commission was I think the icing on the cake. Its significance, I think, lay far more in its relation to Squires and his activities than in the fact of the Royal Commission as such.
87. The allegations made against the appellant in the Royal Commission did emanate from his transactions with Squires. Dr Canaris postulates that the significance of being mentioned in that venue was that it re-inforced the appellant's perception that he was vulnerable to attack by Squires on many fronts and not just to physical violence.
88. Dr Hill, psychiatrist, seeing the appellant on 19/5/99 at the instance of the State Authorities Superannuation Board, doesn't actually formulate a concise opinion on the genesis of the depression but clearly regards it as arising from employment circumstances. He had recorded the gun locker event, the mention in the Royal Commission (referred to as ICAC), exposure to mutilated bodies, harassment and threats to kill or "set him up". He concludes that the appellant had suffered a major depressive disorder though not clinically depressed at the time of examination but having a realistic anxiety of possible attack by certain criminals. His view of nexus to employment is clear in his prognostic comment:
In my view he would be psychologically challenged if he returned to his duties resulting in a return of depressive symptoms of a Major Depressive Disorder.
Dr Hill is the sole psychiatrist aware of the appellant's pulmonary condition contemporaneously with his continuing service in the police force and in discussing return to work comments:
[9]
I know he is considerably physically handicapped by the presence of emphysema and employment demanding physical effort would not be tolerated
[10]
The doctor had access to a report of Professor Bryant dated 14/8/97. That report is not in evidence. In the prior March Dr Bryant had reported the appellant's air flow reduced to 47% of predicted normal. Presumably the report available to Dr Hill did not paint a rosy picture of the appellant's pulmonary condition.
89. The appellant was seen by Dr Smith, psychiatrist, at the instance of the respondent on 8/8/00. He records the gamut of events and circumstances involving the appellant's employment. His ultimate summary is encapsulated in his report at page 6:
It is my opinion, that his naming or involvement with the Royal Commission and potential fears of harm by Mr Squires and others and the lengthy period before the matter was clarified were sufficient stressors that did produce psychiatric turmoil of the type I would regard as an Adjustment Disorder with depressed and anxious mood. …On a balance of medical probability however he was exposed to significant emotional stress due to a combination of fears of harm and challenges to his professional integrity. This is predominantly a result of allegations made against him.
90. The ongoing effects of the diagnosis of "Major depressive disorder in remission" are adverted to by several of the specialist psychiatrists. The view of Dr Hill is noted at [88] above. Dr Canaris shares that view:
I cannot see your client ever returning to policing. Although my diagnosis differs somewhat from that on his infirmity certificate, I don't think this is a matter worth disputing. I certainly cannot see factors outside his police service contributing in any substantial way to his illness.
His ultimate comment on the matter is:
It is a moot point whether a man in his 50's who has suffered a very major psychiatric disorder is likely to be able to return to the workforce in the foreseeable future.
91. The contrary view of Dr Subhas is noted at [9] above. The appellant worked on for going on 18 months after the consultation with Dr Lewin so it was not then a particularly relevant subject. Dr Smith gives his opinion very tersely:
From a practical point of view, his ability to serve with the NSW Police Service in his former role would not be practicable.
Emphysema as a factor in depression
92. The virtually unmentioned element in regard to the aetiology of the depressive illness is the appellant's intercurrent pulmonary disease. This was a chronic and progressively life threatening condition which ultimately, after retirement, required total lung transplantations. It is a condition which one could have contemplated as having depressing effects on the sufferer.
93. The comments of Professor Bryant regarding the appellant's psychological reaction to the condition and proposed treatment have been cited at [24], [25] and [28] above. The comments of Dr Subhas cited at [69] confirm such a reaction. The appellant in evidence conceded that he was distressed at various times by the disability and limitations and by the daunting prognostic comments of the doctors. He even conceded that it may have been a factor in his depression.
94. The one psychiatrist who was aware at a fairly critical time before retirement of the severity of the appellant's pulmonary condition was Dr Hill and he is patently aware of fairly grave physical limitations resulting from that condition as mentioned at [88] above. Notwithstanding that he still doesn't appear to have regarded that intercurrent condition as aetiologically relevant to the depressive illness. That is perhaps more impressive that DrSubhas discounting any relevance of the emphysema when he ultimately appears to become aware of it after the appellant's medical retirement and but shortly before fairly heroic surgery.
95. Overall, I would feel that the progressive lung condition was an element in the genesis of the certified psychiatric disorder, even though rather a minor factor.
Conclusions re causes of the emphysema and asthma
96. In my view the major factor in development by the appellant of emphysema was the established alpha-1 antitrypsin deficiency. Of itself it could account for the entire condition. The appellant's personal smoking contributed to the emphysema but not the asthma. I do not accept that his personal smoking was employment related.
97. The deficient air conditioning at the Springwood police station, which certainly existed, contributed to neither. I do not accept the ex cathedra opinion of Dr Burns that it affected the asthmatic component or certainly not in any material way. As Professor Bryant indicates there was no indication of any allergic diathesis nor any account of intercurrent chest infections. The reaction of the appellant to house dust mite on testing by Dr Burns to a standard battery of allergens seems irrelevant the appellant making no complaints of any symptoms in any situations where he may have been exposed to that stimulus. He gave an account to Professor Bryant of being unaware of any allergy to anything either at home or at work.
98. The appellant's intercurrent psychiatric condition also played no part in the evolution of either the emphysema or the asthma. I accept the evidence of Professor Bryant on that matter in regard to the emphysema. Dr Burns concurred in that view. I find the assertion of Dr Burns that psychological factors would affect the asthma as unconvincing in the absence of any explanation of why it would be so.
99. I view the appellant's exposure to passive smoking in the course of his employment after he ceased smoking himself in 1987 as quantitatively small, being quite intermittent and mostly on occasions when sharing a police vehicle with an officer who smoked and did so at the time. The appellant has deposed to at least one officer who refrained from smoking in a police vehicle while the appellant was present. I would think it likely that the appellant was so exposed, probably in much the same degree, in the non-work situations of daily life. I accept the evidence of Professor Bryant and Dr Burns that passive smoking can aggravate established emphysema. In general I agree with Dr Burns that the effects of passive smoking would be dose related - lesser exposure would have lesser effect, greater exposure would greater effect. The comparative dose in the course of employment seems relatively small. Having regard to the very indefinite evidence of the degree of such exposure either at or away from work I agree with Professor Bryant that any effects of such exposure as there was would be "insignificant" and make no clinical difference to the progress of the condition. I do accept, since there was some rather minimal work exposure, that there could be a somewhat minuscule contribution to the emphysema.
Conclusions re causes of the Major depressive disorder in remission
100. The naming at the Royal Commission looms very large when the appellant first consults Dr Subhas. It remained the principal event inducing the depression in that doctor's view. The appellant's account in evidence makes it clear that he was considerably upset when made aware of what had occurred. Dr Lewin. Dr Smith and Dr Canaris all conceive it, in one way or another, as relevant in the genesis of the depressive illness.
101. Dr Lewin notes regarding the naming at the Royal Commission that:
He became very distressed in the aftermath and was quite markedly fearful for his safety.
He goes on to record a history that in the appellant's belief the mention in that venue was orchestrated by Squires. That would explain the fear element on which Dr Lewin places some emphasis. In this he has much in common with the analysis of Dr Canaris who rather discounts the mentioning before the Commission per se and stresses the element of fear generated by what the appellant conceives as a ploy by Squires to "set him up" as he had threatened to do in the past.
102. DrSmith also implicates this event and he too joins it with the appellant's fear for his personal safety. Dr Subhas also places some stress on the appellant's fear of reprisals by Squires and others. He also patently regarded the threats made to the appellant as quite relevant in suggesting that the best thing the police force could do for the appellant was to take those threats seriously and investigate them. That therapeutic advice connotes a view that the threats were a matter of importance in the genesis of the depression.
103. If, as appears the case, the allegations of WS 14 were untrue and implicated the appellant as involved in corruption one could understand the appellant's immediate distress on being made aware of what had transpired. That would be a perfectly normal reaction. Being innocent of alleged misdoings would usually be expected to provoke anger when the allegations are made rather than depression unless one felt that evil forces were at work and innocence might not be an efficacious defence.
104. I believe the anxiety and concern long felt by the applicant in regard to these threats crystallised with the allegations being made public and that this was the substantial factor in the onset of the symptoms complained of by the appellant immediately following his awareness of what had transpired. He saw it as a manifestation of Squires proclaimed aim to set him up and feared that it could be successful. This over the ensuing months was the substantial factor is his ultimate decompensation.
105. Having decided that the appellant's employment was or could be a factor, in markedly varying degree, to the evolution of various elements of the infirmities determined by PSAC it became apparent that whether the Commissioner's determination that the conditions did not result from the appellant being hurt on duty was correct could depend upon whether or not the requisite nexus existed. That could well depend upon the categorisation of the injury in terms of s4 WCA, the date of injury and the then applicable law.
Injury and nexus
106. This was not an area dealt with to any substantial extent in prior oral or written submissions of the parties. Since it appeared potentially quite critical to the resolution of this appeal I sent a note to counsel for each party on 5 January 2001 raising six questions on which I would seek their further assistance. These questions were:
1 Are the conditions suffered by Reed diseases?
2 If so, when did he receive injury within s 16?
3 Is the date of injury the same in respect of all components?
4 Which portion of the definition in s 4 applies in respect of each component?
5 If the date of injury in any case is after 9/1/97 does s 9A apply?
6 If so, in respect of which element/s of the claim?
The last submission in response to that inquiry was received on 14 February 2001.
107. I had also inquired of counsel in regard to a prior claim for HOD benefits by the appellant in respect of that lengthy absence because of depression in 1996 -97. Jurisdiction to determine any dispute regarding that claim was vested in the Industrial Relations Commission. I was concerned that, perhaps, such had been determined and thereby give rise to issue estoppels. Both counsel have assured me that any such proceeding, if it exists, has not been determined.
108. Mr Walsh for the appellant has submitted that all elements covered by the certificate of PSAC are diseases. Mr Spartalis for the respondent agrees. Mr Walsh submits that it would be found that the emphysema was a pre-existing disease which had been aggravated by the employment within s 4(b)(ii) WCA but that it was more likely that the asthma component and the depression were diseases contracted in the course of the employment within s 4(b)(i) WCA. Mr Spartalis suggests all would fall within s 4(b)(ii) WCA, if injury at all, with the concession that, perhaps, it could be found that the depressive element was contracted in the course of the employment within s 4(b)(i) WCA.
109. For the purposes of determining the date of injury both sections 15 and 16 provide identical bases. It is therefore immaterial for that purpose whether any particular element of the appellant's infirmities is either contracted in the course of the employment or was a pre-existing condition aggravated by the employment.
110. In their supplementary written submissions both parties have suggested that the last day of employment would be the deemed date of injury. It is unclear as to when the appellant actually ceased active work. Dr Canaris and Dr Hill both record a history of him not working since October 1998. The sick leave records forming part of Ex 4 suggest that the appellant continued to work after that time. Those records show absences in relation to the condition of emphysema on 8/10/98 and from 15/10/98 to 20/10/98 and lastly from 1/3/99 to 20/3/99. The precise date is legally inconsequential the applicable law being the same at any potentially relevant time.
111. In the context of this claim the application for medical discharge would be the nearest equivalent of a claim for compensation since this appeal arises from the consequent medical discharge and the determination of its aetiology. That application is dated 20/10/98. For all relevant purposes that would appear to be the deemed date of injury within s15 or s 16.
The application of Section 9A (Part 1)
112. That date, or any likely variant, being after 12 January 1997 s9A potentially requires the appellant to establish that the employment was a substantial contributing factor to the contracting of, or aggravation of, the pulmonary condition. The word "aggravation" is used in this judgment, except where clearly differentiated, to compendiously include the concepts of acceleration, exacerbation and deterioration.
113. Mr Walsh is quite antipathetic to any suggestion that the application of s9A could be invoked in this matter. This he made clear from the time the subject was first canvassed in address by the respondent. He emphasises that at the heel of the hunt, in addresses, the respondent sought and was given leave, to amend the answer filed to include reliance upon s11A and s14 as defences. (I might interpolate that neither matter was ever raised again). But at no time did the respondent seek to amend to rely upon s9A. This is undoubtedly true. The answer filed does not rely upon that provision as any defence to this appeal and leave has not been sought to amend to do so.
114. CCR Part 11, r. 3 provides, inter alia:
(1) A respondent to proceedings may, not later than 28 days after service on him of the originating process in the proceedings, file, and serve on all other parties to the proceedings, an answer setting out "
(a) any grounds on which he intends to deny wholly or partially the applicant's claim
Any defence relying upon s9A would certainly fall within the ambit of the rule and, not being raised, prima facie should not require determination.
[11]
The failure to plead the defence is a prima facie waiver. Waiver is a rather "ornery beast". It is discussed at considerable length by the High Court in The Commonwealth v Verwayen (1990) 170 CLR 394 and more recently by the Court of Appeal in North Broken Hill v Vockins (1999) 19 NSWCCR 193. The former concerned a defence under the Limitation Act, at least now but perhaps not then, regarded as a substantive rather than procedural matter - (vide John Pfieffer P/L v Rogerson [2000] HCA 36) - and the latter compliance with the former s106E WCA, a procedural matter. The provisions of s9A I would regard as substantive in nature.
In regard to the psychiatric condition any purported defence relying upon s9A seems fruitless. The only possible non-employment factor suggested by the respondent is the involvement with the Royal Commission it being submitted that such was an event independent of the employment. For reasons set out above [99] to [104] I regard that involvement as intimately connected with the employment. No other non-employment factor has been suggested. In any event the major element inducing the depression was the fear of reprisals by Squires. Of itself that would satisfy the requirements of s9A.
In regard to the asthma condition reliance upon s9A is unnecessary. I would not be satisfied that the employment was a contributing factor in the genesis of that condition let alone a substantial contributing factor. Dr Burns assumes an allergic diathesis evidenced by the positive response to house dust mite. The applicant's twenty years of personal smoking did not appear to have induced any symptoms of asthma. In so far as it could appear that his reaction to passive smoking in latter years was compatible with asthma I would be satisfied on the evidence that it was mere revelation of any underlying allergic diathesis and not a factor which aggravated, other than very transiently, the asthma condition.
It is in the context of the condition of emphysema that the application or otherwise of s9A could be critical. I have determined above [98] that the only potentially relevant employment factor was exposure to passive smoking in the course of the employment. I accept the evidence of Professor Bryant such exposure could reduce lung function by between 1% and 3%. Neither he nor Dr Burns, who was clearly of the same opinion, recorded any history as to the degree of exposure to passive smoking. The effects of passive smoking would depend upon the degree of exposure - Dr Burns has affirmed that. The appellant had relatively small employment exposure. The decrement in lung function thereby induced, if any, would therefore have been at the lower end of the suggested range and probably even less.
[12]
The question now posed is whether the clinically and symptomatically insignificant increase in the degree of emphysema constitutes an "aggravation, acceleration, exacerbation or deterioration" of the appellant's pre-existing pulmonary condition. That brings to mind the words of Windeyer J in Federal Broom Co Pty Ltd v Semlitch (1964) 110 CLR 626 at 639 concerning those terms:
The words have somewhat different meanings: one may be more apt that another to describe the circumstances of a particular case: but their several meanings are not exclusive of one another. The question that each poses is, it seems to me, whether the disease has been made worse in the sense of more grave, more grievous or more serious in its effects upon the patient.
Kitto J (with whom Taylor and Owen JJ concurred) at 634 appears to follow the same path albeit differently expressed:
The four substantives are not synonymous with each other and a court should assume that it is for the differing shades of meaning of which they are susceptible that the draftsman has chosen to employ them all. They are not all given their true force by asking simply whether the disease has been made worse.
Of the word "exacerbation" he echoed the opinion of Windeyer J when he said:
As applied to a disease it is properly used to refer to effects which the disease produces in the victim rather than the advance of the disease itself to a more serious stage of its development.
Kitto J also cited with approval the comment of Moffitt J in the Full Court of the Supreme Court of New South Wales, the decision in that Court being the subject of the appeal:
There is an exacerbation of a disease where the experience of the disease by the patient is increased or intensified by an increase or intensifying of symptoms. The word is directed to the individual and the effect of the disease upon him rather than being concerned with the underlying mechanism.
In regard to "exacerbation" the Court was unanimous that it was the effects on the sufferer rather than any effect on the disease itself which was the critical determinant.
There is possibly a superficial appearance of conflict between the views of Windeyer and Kitto JJ, the former emphasising a test of whether it is made worse and the latter negating a test of whether it has been made worse. They speak of different "its". The former of the manifestations of a disease and the latter the disease itself. Both agree that it is the effects on the patient that is critical not the effects on the disease. In Semlitch the finding at first instance had been of "exacerbation" of the worker's disease and not any of the other elements of the definition and what passed from Windeyer and Kitto JJ could be said secundum subjectam materiam. Nonetheless, as phrased, each suggests that it is the subjective effects on the person rather than objective effects on the disease that is the ultimate test in respect of the existence of each element of the definition.
[13]
DISCLAIMER - Every effort has been made to comply with suppression orders or statutory provisions prohibiting publication that may apply to this judgment or decision. The onus remains on any person using material in the judgment or decision to ensure that the intended use of that material does not breach any such order or provision. Further enquiries may be directed to the Registry of the Court or Tribunal in which it was generated.
The determination of the Commissioner is amended to specify that the major depressive disorder in remission was caused by the member being hurt on duty. The determination in respect of severe emphysema and asthma stands as not hurt on duty. Costs reserved pending further submissions.
While certainly not discarding the impact of the psychiatric element, for the present, it appears more germane to the major issue of the nexus to employment of the appellant's determined disabilities to consider what I would regard as the major element, the respiratory condition.
The certified "Severe emphysema and asthma"
The applicant has deposed that he was aware of some respiratory symptoms from about 1978 or 1979. Dr Choon Lee seeing the appellant on 23/4/91 had a history of the appellant experiencing some shortness of breath while doing physical exercise around 1979. Dr Bryant, seeing the appellant on 7/11/91, received a history of symptoms commencing some 10 years previously and gradually increasing since though still able to run 100 yards or climb two or three flights of stairs before having to stop. Both doctors were given a history of the appellant ceasing smoking in 1987. The applicant in evidence has insisted that he quit smoking in 1984 or 1985 but his earlier recollection is more probably correct, though not much would turn upon the precise date.
The applicant had been a smoker for about 20 years prior to ceasing. The recorded accounts of the number of cigarettes per day varies. Dr Choon Lee recorded 25-30 per day. In evidence the applicant estimated about 20 per day initially rising to about 25 per day by 1979 when he was stationed at Blacktown. That is within the general range of subsequent accounts. Even after ceasing himself he was still exposed to passive smoking.
Much has been said in evidence about smoking being part of the police "culture". The term is probably used approximately in the sociological sense of "the sum total of ways of living built up by a group of human beings, which is transmitted from one generation to another" (Macquarie Dictionary). The implication is that smoking was a common facet of the lifestyle of the older members of the force and the younger members tended to adopt the habits and attitudes of their more experienced peers. It is hardly an explanation of the appellant's smoking as he was a smoker prior to joining the Police Service starting apparently when he did his apprenticeship in plumbing and continuing thereafter while he worked in the trade.
By 1984 when the appellant was transferred to Parramatta smoking had been officially banned, though apparently the ban was not universally observed. This appears particularly to be so in interview rooms and meal rooms and also in police motor vehicles and in the precincts of courthouses.
The earliest medical material in evidence is an x-ray report of Dr Bell of 2/7/87. The report is addressed to Dr Khan the appellant's long term general practitioner at Springwood where he had lived for some 20 years. It reports that chest x-ray shows:
Gross changes of chronic obstructive airway disease are present in both lungs. No recent lung lesion seen. The heart is not enlarged.
"Chronic obstructive airways disease" for all present practical purposes is synonymous with "emphysema".
The appellant has not introduced into evidence any material from Dr Khan (other than a terse referral note to Dr Subhas on 24/4/96). It would be the likely source of the earliest recorded complaints of the appellant regarding his respiratory problem and the likely reliable source of the history of its evolution. It was Dr Khan who referred the appellant to Dr Choon Lee, consultant physician, in early 1991 who was the first specialist consulted as far as can be determined on the evidence. The reports of Dr Choon Lee were introduced into evidence in the respondent's case - as were the slightly later reports of Professor Bryant who had the oversight of the appellant's treatment from later in 1991 to the present. Neither of the specialists are of any substantial assistance to the appellant on the physical component. One could infer that Dr Khan was similarly unhelpful - and also on the psychiatric component. However any such inference could be unfair. I note on the court matter information sheet that multiple subpoenas were directed to Dr Khan and no records were produced in response thereto.
On 23/4/91 Dr Choon Lee recorded that clinically there was mild shortness of breath on dressing and undressing quickly and a shade of central cyanosis. He organised a series of relevant tests. These revealed anomalies which induced the doctor to believe that even at that stage home oxygen therapy should be beneficial and suggest a prognosis of possible lung transplant in the medium to long term. He particularly noted an Alpha 1 antitrypsin deficiency. He apparently had access to the report (Ex 25) showing alpha-1 antitrypsin of 305 mg/L against a normal range of 1440-2710 mg/L. On 24/6/91 Dr Choon Lee reported to Dr Khan that the appellant had severe emphysema with little reversibility and that the future was of inexorable progression. He encouraged the appellant to cease his employment and counselled against physical activity.
Professor Bryant first saw the appellant on 7/7/91. He noted that the appellant had no symptoms of bronchitis and rarely suffered chest infections. The appellant was unaware of any work exposure to pulmonary irritants. He noted radiographic features of emphysema and that spirometry showed moderately severe air flow limitation (FEV1 2.2 litres) of 52% of predicted normal. He went on to comment:
Any discussion of transplantation with this man is premature because of the significant preservation of lung function. I explained to him that it is difficult to predict the rate of which emphysema due to alpha1 antitrypsin deficiency will progress but, if he stops smoking and has chest infections rigorously controlled, it is quite possible that ten to fifteen years or more could go by before active consideration would need to be given to such a procedure.
The alpha1 antitrypsin deficiency being a genetically inherited condition the professor commended testing of the appellant's siblings and children.
Even though PSAC had certified that the appellant also suffered asthma Professor Bryant was never convinced of the diagnosis. He commented in the report of 15/12/98:
He has no evidence of any co-existent asthma and there is no significant reversibility of his air flow limitation following the use of bronchodilator sprays and there is no significant improvement in his pulmonary function with high doses of oral corticosteroids. He gives no history of any allergic reactions and there is no history of any significant exposure to dust or irritating fumes either at work or at home.
The professor also commented on one of the appellant's alleged employment aggravating factors:
Mr Reed feels that exposure to the air conditioning system at the place of his previous work may have contributed to his condition. He believes this may have happened because of widely fluctuating temperature levels within his office. I do not believe that there is any evidence that temperature fluctuations are directly harmful to patients with emphysema. However, it is possible that such temperature fluctuations could increase the likelihood of him developing coryzal illnesses or lower respiratory tract infections. However, I have no evidence that this was a problem in his case. The fundamental cause for his emphysema is Alpha-1 antitrypsin deficiency and previous cigarette smoking.
In a letter dated 11/4/00 directed to the appellant's solicitors, who patently sought comment of the nexus between depression and aggravation of emphysema, he offered this opinion:
I have no direct knowledge of a relationship between Mr Reed's emphysema and "recurrent depression, personality disorder and paranoia". However Mr Reed has told me on a number of occasions that he was depressed because of his awareness of his impaired lung function and the restrictions that this was placing on his current activities and of his fears that his life would be shortened because of his medical condition. I believe that his fears in this regard were well founded.
Under date of 20/10/00 the professor gives his final comment on the proposed diagnosis of asthma. By this time the appellant has undergone transplantation and Professor Bryant has access to post-surgery pathology of the excised lungs which showed no evidence of any underlying asthma. He also adds his final comments on the causes of the appellant's emphysema:
It is my belief that his cigarette smoking, in association with an alpha-1 antitrypsin deficiency, resulted in his developing emphysema. Current evidence supports the view that between 10% and 15% of active smokers will develop emphysema and that smoking is the single most important cause of emphysema with alpha-1 antitrypsin deficiency being the next most important cause.
Alpha-1 antitrypsin deficiency occurs in between 2% and 3% of the population and, of these, between 75% and 85% will develop emphysema. The likelihood of emphysema occurring is substantially higher in smokers who have the deficiency compared to non-smokers.
The effects of passive smoking have been the subject of widespread interest in recent years. However few studies have examined the impact of passive smoking on respiratory symptom frequency in adults and on rate of decline of lung function. The American Thoracic Society in their position paper "Cigarette smoking and health" reached the conclusion that passive smoking is associated with only modest declines in levels of lung function (between 1% and 3%) which are unlikely to be of clinical significance. I therefore take the view that the emphysema which Mr Reed has developed has been caused both by his active cigarette smoking and by his alpha-1 antitrypsin deficiency. It is my belief that passive smoking has contributed to his developing emphysema and to his deterioration by only a small and insignificant amount (that is less than 3%).
Dr Burns, a thoracic specialist, had been qualified to give evidence in the appellant's case. He saw the appellant on 17/5/00. His history is a little at variance with that recorded above. He notes that the appellant smoked from his youth to about 15 years ago. Not remarkably different - it was more likely until 13 years ago. He was informed the appellant did a PT course in 1979 for three months and topped the course. Inferentially he understands the appellant to have then had no respiratory symptoms. Dr Choon Lee in 1991 had recorded that the appellant on the occasion of this PT course being aware of some breathlessness. He notes that Professor Bryant in 1997 found no evidence of alpha-1 antitrypsin deficiency - that is not the fact; quite the contrary is true. He also records that the appellant has not seen Professor Bryant since 1997 which is far from the fact. He had a history of the appellant reacting by coughing and getting a sensation of chest tightness to exposure to cigarette smoke for years. He also had an account of the variable performance of the air conditioning at the Springwood police station and the appellant's view that such aggravated his symptoms. He noted that the appellant had become increasingly breathless with the passage of time. The doctor also had access to some tests which he arranged.
Dr Burns comments:
Mr Reed has severe emphysema. This, in my experience, is unusual at the age of 50 in the absence of congenital abnormality of alpha-1 antitrypsin deficiency, but it does occur and with our present knowledge, the only factor that we can attribute to the emphysema is smoking. In his case, superimposed on the emphysema is asthma. This is indicated by the positive allergy tests. Generally, airway obstruction in the presence of allergy means that asthma is present and there is some improvement in his FEV1 with bronchodilator inhalation. I believe therefore that he has two conditions. The one is aggravating the other.
Dr Burns is patently unaware of the established alpha-1 antitrypsin deficiency and therefore cannot factor that matter into the aetiological equation. His reliance on allergy tests seems to be drawing a long bow. The doctor reports that the appellant was submitted to a standard battery of allergens and showed a positive reaction only to house dust mite.
DrBurns also thought that the inefficient air conditioning at Springwood would likely aggravate the asthmatic component which he regarded as a significant element of the appellant's respiratory problem though not of the same order as the emphysema. That matter would not be a factor in aggravation of the emphysema. The asthma would account for a degree of the airways obstruction experienced by the appellant.
In a supplementary report dated 25/10/00 the doctor clarifies a comment in the original report to indicate that he intended to communicate that both physical and psychological (rather than physiological) stress would exacerbate asthma. He also considered passive smoking as a contributor to the emphysema:
It is likely that his smoking habit between 1974 and 1994 materially caused and accelerated his emphysema. Not as much is known about the effect of passive smoking on emphysema as active smoking, but the effect of cigarette smoking on causing emphysema is likely to be dose related. One's own cigarette smoking is therefore likely to produce more damage than the lower dose inhaled in passive smoking. However, it is likely that the latter would act in the same way as personal smoking and add to the amount of irritation and hence would materially aggravate the emphysema. I believe that this did happen in his case, on the balance of probabilities.
The expressed period of smoking is clearly wrong. I believe the doctor intended to communicate a 20 year span and probably, on the history that he had, commencing in youth and concluding in 1984 rather than 1994.
Dr George Hall saw the appellant on 25/6/99 to advise the State Authorities Superannuation Board. He noted that the appellant was obviously breathless at rest, with audible breathing and some wheezing. He was cyanosed; possibly some clubbing of the fingers and a chest expansion of 1.5 inches. He noted some information from Professor Bryant, particularly an FEV 1 of 1.55 litres - 41% of predicted normal. He thought the applicant essentially totally and permanently incapacitated by emphysema and asthma. He gave no consideration to the aetiology of the conditions.
It must be accepted that the appellant suffered from emphysema and asthma. PSAC has so certified and the certificate of PSAC is beyond challenge. Of course, it is highly improbable that the appellant now suffers either complaint. Presumably the lung transplant has remedied both defects. I would assume that the transplants were non-emphysematous. What the certificate of PSAC does not conclude is the degree to which the appellant suffered asthma. It does describe the emphysema as severe - and it clearly was - but there is no such descriptive adjective regarding the asthma. Using the external evidence of the specialists to interpret the phrase "severe emphysema and asthma" I would feel that the adjective severe refers only to the emphysema element. In the light of the evidence of Professor Bryant the asthma appears to have been a minor element of the appellant's respiratory problems. DrBurns takes a different view but I regard his opinions as seriously flawed. He assumed that the appellant had no alpha-1 antitrypsin deficiency. That is clearly wrong. Professor Bryant and DrChoon Lee make it clear that that condition not only existed but was the major factor in the development of the emphysema. Being unaware of that factor Dr Burns was in the invidious position of endeavouring to explain the evolution of the appellant's respiratory problems without knowledge of the major contributing factor. It is not surprising that he invoked other factors as substantial contributors in compensation for that deficit in his knowledge.
The evidence for asthma of any significant degree is scant. Dr Burns exposed the appellant to a battery of standard allergens with a positive response only to house dust mite. Professor Bryant, as Dr Choon Lee before him, makes it clear that there was no significant reversal of the appellant's air flow limitations with the administration of broncho-dilators and ingestion of corticosteroids. Such a reversal would be expected where asthma is a significant factor. Professor Bryant also noted, post-surgery, that there was no pathological evidence of asthma in the removed lungs. Therefore, while accepting that the appellant suffered asthma, I do not regard such condition as playing any material part in his overall respiratory condition. Emphysema was the overwhelming cause of the respiratory symptoms.
I accept Professor Bryant's expert evidence. Given the appellant's genetic alpha-1 antitrypsin deficiency there was, without any other factor, a 75% to 85% chance that he would develop emphysema. Active smoking would increase the risk of developing the condition at least within that range - probably, I gather, even beyond the top of that range. Even without the genetic deficiency Professor Bryant indicates that 10% - 15% of active smokers will develop emphysema. Emphysema having initiated, as Dr Choon Lee indicated on 24/6/91, the prognosis was of inexorable progression - at least in the presence of the established alpha 1 antitrypsin deficiency. That is implicit in the evidence of Professor Bryant also.
Personal smoking
The evidence is convincing that the fundamental causes of the appellant's emphysema is the genetic deficiency and the active smoking for 20 years. Given that situation the appellant submits, rather indirectly, that the appellant's personal smoking was a facet of his employment and, more directly, as was exposure to passive smoking, the effects of the defective air conditioning at Springwood and the impact of the psychological disturbance emanating from employment experiences. A number of those factors are more directed to a nexus with the intercurrent asthma.
I do not accept the implicit suggestion that the appellant's personal smoking was an employment related activity, that it was something induced by his employment. The appellant has cited the oft quoted passage of Dixon J in Humphrey Earl Ltd v Speechley (1951) 84 CLR 126 at 133:
The service is not confined to the actual performance of the work which the workman is employed to do. Whatever is incidental to the performance of the work is covered by the course of the employment. When an accident occurs in intervals between work the question whether it occurs in the course of the employment must depend upon the answer to the question whether the workman was doing something which he was reasonably required, expected or authorised to do in order to carry out his duties.
Hatzimanolis v ANI Corporation Limited (1992) 173 CLR 473 and (1992) 8 NSWCCR 242; Cudgegong Soaring P/L v Harris (1996) 13 NSWCCR 92 and WorkCover Authority v Billpatt Holdings P/L (1995) 11 NSWCCR 565 have explicated the use of the word "authorised" in the citation from Dixon J. They place some stress upon an employer not merely passively permitting a particular activity but actively encouraging or inducing it before the requisite nexus to employment can be established. It is not so much that an employer did little, if anything, to preclude a particular activity but whether he did something positive towards encouraging it.
At least by the time that the appellant was transferred to the Ferguson Centre at Parramatta in 1984 he has indicated that smoking was "banned" by the employer. The Ferguson Centre was a focus of some of the appellant's major complaints in this regard. The employer was hardly encouraging or inducing the particular activity it was rather to the contrary. The applicant, on the histories given to Dr Choon Lee and Professor Bryant in 1991, which I accept as the more accurate account, continued to smoke himself for some few years after 1984.
The appellant suggests that smoking functioned as a pacifier or relaxant. The appellant smoked before entering the police force. He was presumably smoking for much the same reasons before and after. It was not a means adopted to deal with perceived problems arising from the employment but rather a developed habit that continued into the employment. It was an addiction personal to the appellant, harmful to health and long known to be so. He could have been addicted to alcohol or narcotics. An existing addiction per se does not arise out of an employment. Nor does the fact that it is persisted in while working make it an integral part of the employment such that any consequences of it occur relevantly as part of the employment.
It has been suggested that smoking was a part of the police culture. Perhaps policemen smoke relatively more than those in other occupations. Perhaps they don't. No evidence has been directed to that question. I do not regard the fact, if it be so, that many officers smoked renders smoking ancillary to the employment.
On the evidence it appears that the appellant smoked both while working and while not because he wished to. It is not suggested in evidence that he smoked in any different degree at week-ends away from work than during work time. It was something that the appellant did independent of the work situation as well as while working. Nothing gave it any particular employment characteristic.
I do not regard any consequence of the appellant's personal smoking as falling within the category of injury arising out of or in the course of the employment.
Passive smoking
Passive smoking is another matter. If smoking occurs in an enclosed area both smokers and non-smokers are exposed to the contaminated air. If that occurs in an employment situation it becomes but part of the work environment. During the period from 1987 when the appellant himself ceased to smoke he was attached to the Major Crime Squad and stationed at the Ferguson Centre at Parramatta until 1994. He was then transferred to Springwood as Chief of Detectives.
The evidence does not suggest any great degree of exposure during this period. Apparently there was some continued smoking in interview rooms and meal rooms and certainly in police motor vehicles. Indeed, the appellant deposed that most of the exposure was in police motor vehicles. Dr Burns has suggested that the effects of passive smoking are probably dose-related. That does not appear incompatible in principle with the evidence of Professor Bryant. In effect the larger dose of personal smoking would be more harmful than the much smaller dose involved in passive smoking.
The problem on the evidence is to make any sensible evaluation of the dosage involved from passive smoking as far as the appellant is concerned. The exposure in the work environment was clearly intermittent. There is no evidence as to the frequency with which the appellant shared police vehicles with officers who were smokers and did smoke in those vehicles or shared interview rooms with such officers. It is all very amorphous. All that can be said really is that were occasions when the appellant was so exposed. When such occurred he has deposed that he would get tight in the chest and feel ill. All that can be definitively said is that there were occasions when the appellant was exposed to passive smoking in the workplace. It was probably no less so elsewhere when away from the employment.
Both the thoracic specialists, Dr Burns and Professor Bryant, agree that passive smoking can aggravate or contribute to emphysema. Neither indicate the assumed degree of exposure upon which their particular opinions in this case were founded. On whatever assumed degree Dr Burns considers the effects "material" and Professor Bryant "insignificant". One would assume that the work environment was probably not the only circumstance in which the appellant was so exposed. In that case whatever the effects of passive smoking it was not solely an employment factor.
On the totality of the evidence exposure to passive smoking in the workplace could be a factor in the evolution of the appellant's emphysema though the degree of such aggravation, if any, was probably, as Professor Bryant phrases it, clinically "insignificant" in the overall picture. This is a topic to which I will return later in these reasons at [118].
Defective air conditioning
In the period from 1994 when the appellant was stationed at Springwood the air conditioning was quite ineffective providing major swings in ambient temperature. Professor Bryant finds this factor irrelevant to the emphysema in the absence of chest infections. He had no history, or clinical awareness, of any such consequence and so dismisses it as relevant to the development of emphysema. In that he appears to have the assent of Dr Burns as far as emphysema is concerned. In his report of 18/5/00 Dr Burns comments:
However, from his description of his work in the Springwood Police Station, particularly in those years from 1994 to 1998, it is likely that the environmental fluctuations and probably the poorly serviced air conditioner aggravated the asthmatic component of his chest disease.
Professor Bryant doesn't canvass the contribution of the Springwood environmental situation to the asthma component since he does not believe that such a component exists. His reasons for so concluding are rather compelling. However PSAC maintains that there is such a component and PSAC is incontrovertible.
Dr Burns gives no account of why such would aggravate the asthmatic component. It is just a bland statement of conclusion by an expert. There is no reference to any medical principles whereby the Court could itself apply such principles to the facts as it finds them to determine the nexus itself. The statement falls within the category of "oracular pronouncements of experts" rather deprecated by Lord President Cooper in Davie v Edinburgh Magistrates 1953 SC 34 where he added apropos the expert:
Their duty is to furnish the judge or jury with the necessary scientific criteria for testing the accuracy of their conclusions, so as to enable the judge or jury to form their own independent judgment by the application of these criteria to the facts proved in evidence.
While there is an asthmatic component to the appellant's respiratory disease it is, on the totality of the evidence, of minimal degree and even if there were a degree of manifestation of symptoms due to variation of ambient temperatures it seems more likely to be mere transient manifestation of an existing problem than any ongoing exacerbation of the asthmatic condition.
Psychological stress
As will be discussed a little later in these reasons the appellant was psychologically affected by various experiences some of which at least have a requisite work nexus. The question here considered is whether such experiences impinged upon the respiratory condition.
The comment of Dr Burns on this aspect in his report of 18/5/00, suitably amended by his subsequent report of 25/10/00, is:
In recent years he has become more short of breath than ever. Physical and psychological (not physiological as in the initial report) stress would produce a sensation of chest tightness.
This would appear to relate to transient symptoms rather than continuing deterioration of the underlying condition.
Professor Bryant in the passage quoted in [29] above sees no impact of psychological stress on the respiratory problem as such. Dr Burn's comments are not inconsistent with such a view. In my view any psychological stress has not contributed to the evolution of the appellant's emphysema and asthma, though the reverse may be true.
The certified "Major depressive disorder in remission"
The first relevant evidence emanates from Dr Khan on 24/4/96 when he referred the appellant to Dr Subhas, psychiatrist, in a very terse note:
Herewith Raymond Reed with stress symptoms for your kind evaluation & management/advice.
It was similarly tersely noted that there was a past history of "COAD/Asthma; anxiety & depression". Over just how long that "past history" extended would have been interesting to know, particularly, in the present context, any past anxiety and/or depression.
The appellant was first seen by Dr Subhas on 30/5/96. A diagnosis of Major Depression was made. The doctor noted the factors of which the appellant complained as:
· he had lost trust in his colleagues.
· he was named before the Royal Commission by a policeman, very upsetting.
· angry and humiliated that his name appeared in the Sydney Morning Herald in regard to that "naming".
· he felt his life was under threat and was harassed by criminals.
· Internal Affairs had ignored his reports of threatening phone calls and such like.
By 29/8/96 he reported being more disillusioned and disappointed with the Police Force and feeling more unsafe than ever. The appellant declined, as he had previously, to follow the doctor's advice to desist from work for a time.
On 22/10/96 the appellant phoned the doctor for an urgent appointment which took place later that day. He complained of a confrontation with one of his colleagues; he expressed concern about his job; he felt he was the target of criminals and was ruminating about things that happened at work. The appellant on this occasion agreed to take time off work and was in fact off from that day for almost six months until 12/4/97 - it may have been until late May (vide report of Dr Lewin, 16/5/97, Ex G, final paragraph). During this period he was treated with Prothiaden, an anti-depressant, (originally prescribed by Dr Khan and continued by Dr Subhas), Thioridazine, an anti-psychotic, used as a tranquilliser, and Prozac, an anti-depressant. By 14/1/97 he was less depressed than he had been. Dr Subhas thought that the Department could best assist Mr Reed by taking seriously and investigating his belief that his life was under threat by particular criminals with whom he had dealt and similarly the false allegations before the Royal Commission.
The appellant has deposed to the incident occurring on 22/10/96. He had told a subordinate that action by him in a particular matter had been improper The subordinate was clearly annoyed and went to the gun locker which was in the appellant's office, withdrew and donned his firearm. The appellant was rather apprehensive and fearful about the behaviour of the subordinate and also about how he himself might react, he having donned his weapon also. He was so distressed that he phoned Dr Khan and, following his advice, Dr Subhas and arranged the urgent appointment.
Following the consultation of 14/1/97 the next consultation on which the doctor reports (in evidence) is that of 11/8/97. The appellant had resumed as Chief of Detectives at Springwood the previous April - or maybe May. Dr Subhas notes that he appears to be coping reasonably well though he reports "a few problems with bronchitis and pneumonia". He concluded the report of 12/8/97 with the opinion:
I'm confident with ongoing support, he should be able to function normally. At this stage, there is no need for me to see him in the near future. I would like to review him in three or four months if he wishes to come back and see me.
There is a fairly clear implication that Dr Subhas at that time thought the appellant basically "out of the woods".
In a compendious report under date of 3/12/97 Dr Subhas re-iterated to the appellant's attorneys much of what is recorded above and added:
Since Mr Reed went back to work I have been able to review him on three occasions and he seems to be coping reasonably well as far as his job is concerned. His depression seems to have lifted completely and he has persevered with the Anti-Depressant Prozac which I feel he should continue.
On the aetiological aspect he commented:
Having seen Mr Reed on a few occasions it was evident that since the allegations were brought against him before the Royal Commission it has had a devastating affect on him. The whole incident seems to have taken away the trust that he had for his colleagues.
While Dr Subhas appears to be speaking of current colleagues the colleague who had implicated the appellant before the Royal Commission had been out of the police force for something of the order of six years. I doubt it would have made much difference to the doctor's opinion even if specifically aware that it was an ex-colleague who was involved rather than a current fellow officer.
Following a consultation on 9/4/98 Dr Subhas reported to the appellant's attorneys under date of 22/4/98. That report included the following observations:
I reviewed Mr Reed on 9th April 1998 and he seems to be coping well with his job considering the problems he has had over the last couple of years. I must say Mr Reed has overcome his depression and he is coping well. He is now back at work. He told me that he was handling his job well and in fact he is as enthusiastic and dedicated as he always has been in the past.
The penultimate report of Dr Subhas in evidence is that of 15/12/99 following a consultation on 18/10/99. The report post-dates the appellant's retirement by several months. He noted that the patient's major depression seemed to be in remission. He also noted that his physical state had deteriorated "as he is more affected by his asthma and he does tend to get very dyspnoeic". While not depressed he still tends to get anxious and uptight. His final relevant comment is:
There is no doubt his physical state also seems to have deteriorated. When one looks at him, a forty-nine year old man, he is almost looking as if he is in his sixties. He has definitely aged in the last few years. One can say that the pressures of work definitely caught up with him.
Were the doctor aware of the gravity of the intercurrent pulmonary condition he may have found it a more ready explanation of the appellant's physical deterioration.
In his ultimate report of 19/7/00, portions of which are cited in [9] above, Dr Subhas deals with a number of questions raised by the appellant's attorneys. There are a number of aetiologically pertinent observations:
From what I gather he has never had any problems prior to all the incidents that happened during the Royal Commission. One can reasonably hypothesise that this man has been under immense pressure and stress related to all the unsubstantiated complaints against him.
And a little later:
When I reviewed him on 12th July 2000 he told me he is waiting for lung transplant and he has been to see the Transplant Team at St. Vincent's Hospital. Understandably, Mr Reed was rather distressed when he spoke about his physical problems and he believes that he is not going to be here for long unless he finds a suitable donor, and he is aware of the risks involved.
These comments seem to indicate that the Royal Commission and its sequelae are seen as the major employment factors in the genesis of the depression. It is also the first occasion on which Dr Subhas is clearly aware of the potentially catastrophic effects of the appellant's pulmonary condition. Previously, in the report dated 17/2/99, he had noted that the appellant "had been affected by his respiratory problems" and that he understood he suffered from bronchitis with emphysema and in the report of 15/12/99 that he had been more affected by asthma. However it is only in this ultimate report that Dr Subhas reveals any awareness of the gravity of the physical problems and then without any apparent account of the long-term evolution of that problem.
Returning to the factors of which the appellant complained to Dr Subhas on first consultation and those which were broadly summarised in [7] above and to which the appellant has more fully deposed in evidence.
Criminal contacts and threats - RTA affair - ICAC - Ombudsman
While stationed at Parramatta in the early 1990's and through until about 1995 at Springwood the applicant had investigated the activities of one Arthur Squires, who operated a building company at Seven Hills, in regard to theft, fraud, possession of firearms, attempted murder and murder. Over four or five years he was instrumental in preferring some dozen charges against Squires who at one stage decamped to the USA. Squires was upset by these events and had threatened Reed that he wouldn't survive, should get out of the force and such like. Reed reported these threats to his superior, Inspector Cranna and sent a briefing note to Internal Affairs but nothing much happened though Inspector Cranna did request police to keep an eye on his domestic home.
Arising out of one of the investigations regarding Squires, one George Slade, a former police officer, allied with Squires alleged before ICAC that Reed when raiding Squires' premises had helped himself to something like $6,000 and some marijuana. This allegation was never formally resolved to the appellant's considerable distress.
Around 1993 as the trial of Squires in relation to a firearms charge approached the appellant had phone calls from one Mike Smith, an associate of Squires, seeking to arrange a meeting. Reed had some apprehensions about Mike Smith and the purpose of any proposed meeting and side-stepped any such arrangement. He was of the view that Squires was quite capable of implementing the threats that he had made.
Previously, around 1983, the appellant was investigating one Thomas O'Connell a respected inhabitant of the Oberon area in respect of receiving. Reed on a visit to Oberon located what he described as a massive quantity of stolen goods. In the aftermath O'Connell convinced Internal Affairs that Reed was involved with him and his associates. Reed was replaced as the investigating officer. That remained so for a couple of years. The appellant was aware that his phone was tapped - he had since seen transcripts - by Internal Affairs. The Ombudsman was involved. He appears to have given a Scots verdict - .unproven. The control of the O'Connell matter was returned to Reed. When he eventually turned up to the trial at Oberon he found the courthouse surrounded by a SWAS team. When he did give evidence in the trial he was in the witness box for four days. His credit was severely attacked on the basis of the various allegations noted above. The whole affair was quite disturbing.
Jim Murray was a "druggie" with AIDS who practised armed robbery. When arrested at Merrylands he threatened to kill every cop involved. There were quite a number, the appellant among them. In respect of the other officers precautionary measures were taken by suppressing access to RTA records of their home addresses. That did not occur in relation to the appellant who later became aware of that fact and while accepting that there are other means for a criminal to ascertain the home address of an officer questions why was he left out, why was he exposed to greater risk than the others.
These are a few examples of events which the appellant regards as worrisome and causing him some distress and anxiety. There were others involving Hudson, Klee and Richardson of a similar nature. They were stand-over men.
The Royal Commission
In March 1996 the appellant was attending a training course for detective-sergeants at the Police Academy, Goulburn. While he was there on 11/3/96, then unbeknown to him, one "WS14" - an former detective-sergeant colleague of the appellant at Parramatta - named him before the Royal Commission into the Police suggesting that he was involved in theft and receiving. On 13/3/96 the Sydney Morning Herald published that fact and identified the appellant. The appellant was shown a copy of the SMH by a fellow detective doing the Goulburn course. He immediately phoned Sydney to ascertain what he could of the matter and what he should do, without much benefit. He faxed a request to give evidence before the Royal Commission. The appellant says he felt ill, unable to sleep, shattered, his career was finished, his fellow officers appeared to avoid him.
In the aftermath of this publication in the Sydney Morning Herald he was upset, distressed and embarrassed. He lived in a small community at Springwood where this would be a topic of local interest. He sought action by his commander, Inspector Cranna, but he also had been named before the Royal Commission and nothing eventuated. He was shortly after advised that he would not be required to give evidence before the Commission. That would appear to suggest that the Commission was rather unimpressed by the veracity of the allegations which were of a rather serious nature.
On 24/4/96 the applicant consulted Dr Khan and was referred to Dr Subhas whom he saw on 30/5/96. As set out in [60] above his major complaints were of distress relating to the Royal Commission and to the threats to his life by criminals and the inactivity of Internal Affairs on that matter. One of the reasons that the appellant declined to follow the advice of Dr Subhas to take time off was that he didn't wish to draw attention to himself in the aftermath of the events at the Commission.
Gruesome events
The appellant had his share of contact with the recently deceased. Death had not infrequently been in circumstances of considerable violence. He mentioned one particularly dismembered body at Glenbrook. Dr Subhas briefly refers to these matters in his report of 29/1/97:
During his service with the Force he has had a few upsetting incidents - seeing bodies which he seems to have handled reasonably well.
Similarly Dr Canaris adverts to these matters at para 35:
The question left hanging at [51] above was whether in fact the exposure to passive smoking had aggravated, accelerated, exacerbated or deteriorated the appellant's emphysema. Both Professor Bryant and Dr Burns express a view that it did. The appellant has deposed to experiencing some discomforting symptoms when, and while, so exposed. On the totality of the evidence I would be satisfied that such exposure did marginally increase the degree of emphysema. I would also be satisfied that the degree of increase was clinically insignificant as deposed by Professor Bryant. By that I take Professor Bryant to hold the view that, while the condition was marginally increased in degree by the effects of such exposure, in the "big picture" of the overall pulmonary condition the practical effects were indeterminate, negligible and inconsequential. In quantitative terms I would take him to suggest, for instance, that the clinical difference between having 45% and 44% of normal lung function is of no practical symptomatic significance. The condition is equally parlous in either case.
In this appeal it is not within my jurisdiction to determine whether any particular injury results in incapacity, losses, need for medical treatment or indeed any of the consequences that might ensue. I can but determine whether the appellant received injury within s4 WCA and the relevant nexus of such injury, or injuries, to the certified infirmities. PSAC has determined that collectively they render the appellant unfit to be a police officer. Whether any particular one of the certified infirmities has that effect is not a matter determinable by this Court. My only interest in the consequences of alleged injury is limited to assessing such as an aid to the determination of whether there was, in fact, a relevant injury.
It is therefore necessary to divert temporarily from consideration of the application or otherwise of s 9A to consider the previous question of to what it is suggested that the employment contributed, substantially or otherwise.
Aggravation, acceleration, exacerbation or deterioration
It was agreed that the criterion of "exacerbation" is the subjective enhancement of symptoms or disability. It is difficult to contemplate that it is otherwise in regard to "aggravation" or "deterioration". All three words, as suggested by Windeyer J, have a common implication of a worsening - in each case a symptomatic worsening. "Acceleration" is the concept where that approach could seem more questionable. Acceleration appears to assume a disease which is progressive and that injury has caused it to progress more quickly than it otherwise would. Even that concept would also seem to assume that such further progression was evidenced by increased symptoms or disability. Indeed, without being accompanied by such it is difficult to conceive that the supposed injury has had any discernible effect on the disease process. If a man feels no worse, suffers no more and copes as well or poorly after a suggested injury as he did before the lack of any such consequence suggests that the "injury" in question has been irrelevant in the natural history of the disease and its concomitants.
An "injury" without consequence is irrelevant in the chain of causality. That seems compatible with the invocation of common sense, experience and value judgment suggested in March v E & MH Stramare Pty Ltd (1991) 171 CLR 506. Such an injury does not meet even the "but for" or "causa sine qua non" tests which themselves would not be determinative.
On the totality of the evidence the appellant's quite limited exposure to passive smoking in the course of his employment played no discernible part in the evolution of the condition of emphysema by way of aggravation, acceleration, exacerbation or deterioration. In that circumstance the appellant did not receive relevant injury.
Lest it be ultimately assessed otherwise it is desirable to return to the question posed by s9A.
The application of Section 9A - (Part 2)
If it be conceived that the employment exposure to passive smoking was a factor in the aggravation, acceleration, exacerbation or deterioration of the appellant's emphysema then there was a relevant injury within s4(b)(ii) WCA and if that injury be conceived as making a de minimis contribution to the progress of the condition then, as the law stood prior to 12 January 1997, the appellant's condition would result from his being hurt on duty: Chalmers v Bank of NSW (1979) WCR 188; Treloar v Australian Telecommunication Commission (1990) 97 ALR 321: Watts Peterson Automotive v Peterson (1994) 10 NSWCCR 653.
Even were that so, and I do not believe it to be so, the employment exposure could not be a substantial factor in the evolution of the disease - the assumption at most could be some minuscule acceleration I would think. However it seems to me that whether that be so or not is not the question raised by s9A - or by s16(1)(b) for that matter. As suggested in Cant v Catholic Schools Office (2000) 20 NSWCCR 88 the provision requires that the relevant employment be a substantial contributing factor to the injury. The injury is the assumed minimal aggravation, acceleration, exacerbation or deterioration of the emphysema. To that injury, ex hypothesi, the only contributing factor is the employment exposure. If that be the only factor it is necessarily substantial in that context.
It therefore appears to me that in the context of an injury falling within s4(b)(ii) WCA the introduction of s9A makes no difference - substantial or otherwise. It would therefore be fruitless to consider allowing any amendment by the respondent to rely upon that provision.
It could transpire that this evaluation of the impact of s9A is erroneous. Whether an amendment were allowed or not could be critical. While not purporting to have considered that matter deeply since no application to amend has formally been made I would be minded to reject such application if made. It appears to me to raise not merely questions of law but matters of fact that could have been addressed in evidence had the appellant been aware that he was required to confront such a defence. This matter was touched upon recently in the Court of Appeal: BW Esler Services v Dulhunty [2000] NSWCA 349 giving judgment on 20 December 2000 particularly by Powell JA at [31].
The all or nothing submission
Mr Spartalis for the Commissioner has submitted that I am constrained to find that the determination of the Commissioner was wholly right or wholly wrong. As a corollary he submits I cannot find that an individual element of the appellant's multiple infirmities is the result of the appellant being hurt or duty but others not. I believe such submission to be misconceived.
The argument advanced appears to be that PSAC has determined multiple conditions and the appellant has been discharged from the service because of those multiple conditions and the Commissioner has determined none result from HOD. This involves considering the subject matter of this appeal to be the appellant's medical retirement and such rights as might ensue therefrom. It isn't. It is simply whether or not the determination of the Commissioner is correct. There is no rule of law or nature that the Commissioner must be entirely right or entirely wrong. He , as anyone else, can be like the curate's egg, good in parts.
The underlying premise upon which the submission is based appears to be that the PRS Act is a rational and efficient means of determining a former officer's right to particular benefits. Such rights are not, other than quite indirectly, determined by this Court. The ultimate determination of this Court may or may not definitively determine the former officer's pecuniary rights under the PRS Act. The Act is not designed to achieve that end.
As in an appeal from any judgment of a Court the judgment does not need to be completely wrong for an appeal to succeed. The primary judge being correct on five out of six matters raised does not constitute a pass mark. On any particular element the primary decision is right or wrong. If wrong on any material matter the appeal succeeds. The Commissioner's determination is in no different position.
The function of this Court is to determine the correctness or otherwise of the Commissioner's determination and, in so far as it may be wrong, to rectify any error. The consequences that flow from any such rectification are not within the present province of this Court.
Conclusion
The Commissioner determined that the infirmities of "Severe emphysema and asthma; Major depressive disorder in remission" were not caused by the appellant being hurt on duty.
For the reasons discussed above I am of the opinion that he was right in relation to the severe emphysema and asthma but wrong in respect of the major depressive disorder in remission. The determination of the Commissioner is therefore amended to specify that the latter condition was caused by the member being hurt on duty.
On the question of costs I will hear submissions of the parties before making any formal order.
MJ Walsh instructed by Oates & Smith appeared for the appellant.
MS Spartalis instructed by Legal Services - NSW Police Service appeared for the respondent.